0:01 If you have a belly sticking out, you
0:04 have a problem because the fat that's in
0:06 the stomach, that's called visceral fat.
0:09 This is very detrimental fat. And that's
0:11 the epidemic that we have today. But the
0:12 only thing that will make you lose that
0:14 fat very quickly.
0:16 Dr. Praep Jam Nadas is a worldleading cardiologist
0:17 cardiologist
0:19 who has treated more than a quarter of a
0:21 million patients with chronic heart disease.
0:21 disease.
0:23 Now he's using his voice to help
0:25 millions more prevent ending up on the
0:26 operating table through simple lifestyle
0:29 techniques. This is crucial. Glucose
0:31 actually is toxic inside the bloodstream
0:33 and the body cause insulin into the
0:35 bloodstream to push glucose out. But
0:38 frequent consumption of carbs, sugar,
0:39 processed foods is causing insulin to
0:41 stay up which can lead to insulin
0:43 resistance and you're going to be more
0:45 prone to heart disease which is the
0:47 number one cause of death all over the
0:48 world right now. But this is where
0:50 fasting comes in because after 12 hours
0:53 you start pulling the fat out. And the
0:55 first place the fat comes out of is
0:56 going to be visceral fat. But with
0:58 modern living, we have lost this
1:01 physiology of fasting. So we'll go into that.
1:01 that.
1:03 And there must be certain things which
1:05 people aren't aware aren't healthy as it
1:06 relates to my cardiovascular health.
1:09 Yes. So I see that people who overly do
1:11 aerobic activity, they end up with more
1:14 coronary artery disease than patients
1:16 who do short sprints and resistance
1:20 exercises. Then there's mold. And almost
1:22 70% of homes these days have some form
1:25 of mold toxicity in them. But also one
1:27 night of bad sleep, you become insulin
1:29 resistant the next day. And there's
1:31 calcium supplements, excessive fruit.
1:32 White rice.
1:33 White rice.
1:34 You'll be surprised how much arsenic
1:36 there's in rice these days.
1:37 I watch a lot of true crime. People kill
1:38 each other with arsenic.
1:40 Yes. And it'll slowly kill you. And
1:43 lastly, this is going to surprise you.
1:44 I see messages all the time in the
1:46 comments section that some of you didn't
1:48 realize you didn't subscribe. So, if you
1:49 could do me a favor and double check if
1:51 you're a subscriber to this channel,
1:52 that would be tremendously appreciated.
1:54 It's the simple. It's the free thing
1:56 that anybody that watches the show
1:57 frequently can do to help us here to
1:59 keep everything going in this show in
2:01 the trajectory it's on. So, please do
2:03 double check if you've subscribed and uh
2:04 thank you so much because in a strange
2:07 way you are you're part of our history
2:08 and you're on this journey with us and I
2:10 appreciate you for that. So, yeah, thank you
2:11 you [Music]
2:14 [Music]
2:16 Dr. Pep Gnardas.
2:18 What is it you spent the entirety of
2:20 your career especially the last few
2:23 decades of your career focusing on
2:26 prevention of heart disease which is the
2:28 number one cause of death all over the
2:30 world right now. You see the heart is
2:32 made up of many parts. So this is
2:35 important. It's a pump. So it's a
2:37 muscle. So you have diseases of the
2:39 muscle and it's rampant these days. Cardiomyopathy,
2:41 Cardiomyopathy,
2:42 weakness of the muscle. Then you have
2:45 the arteries on top of the heart and
2:47 that's called coronary artery disease.
2:49 Diseases of the arteries they block up.
2:51 Then you have the valves. The valves
2:53 tend to get damaged as well. For
2:55 example, aotic stenosis. And then you
2:57 have the peripheral circulation. All the
2:59 blood vessels that go to your corateeds,
3:02 your brain, your legs and to all the
3:04 organs of your body. And then you have
3:08 microvascular disease which is the tiny capillaries
3:10 capillaries
3:12 that they become dysfunctional as well.
3:13 What's a capillary?
3:15 Capillary is the branch of the branch of
3:15 the branch.
3:17 Okay. So like the tiny little veins
3:19 final ones. Yeah. So when they become
3:22 dysfunctional that organ becomes
3:26 dysfunctional as well. So cardiology
3:29 affects all these aspects.
3:31 And how long have you been operating on
3:33 people's hearts? about 35 years now.
3:35 And how many hearts do you think you've treated?
3:37 treated?
3:39 In excess of 30,000.
3:41 And what what's the age range of those patients?
3:43 patients?
3:45 They were used to be mostly older people
3:49 over 65. Now I would say 50% of them are
3:54 under 65 and 50% are over 65. So the
3:56 demographics is changing. The younger
3:58 people are developing heart disease. So,
3:59 the youngest patient you've ever treated?
4:01 treated? 28.
4:02 28.
4:04 And what what was the predicament in
4:05 that case?
4:08 He was having a full-blown heart attack.
4:10 So, that means that one of his arteries
4:11 was completely clogged up. It had a
4:13 blood clot in it. There was no
4:15 circulation past that artery and
4:17 therefore he was having acute chest
4:21 pain, sweating, blood pressure was low.
4:22 He comes straight to the hospital. I
4:24 rushed to the hospital, take him
4:26 straight into the kath lab and try to
4:28 open up that blockage. And we did open
4:30 up that blockage and put a stent in it
4:32 and got rid of that blockage immediately
4:34 to restore that blood flow to the heart muscle.
4:35 muscle.
4:37 And at 28, how does one get a heart
4:39 attack? You said it was blocked up.
4:42 There's a lot of nuance in that. Most
4:44 people don't understand what a heart
4:46 attack is. So, in a nutshell, I'm going
4:48 to tell you what it is. When you have a
4:52 plaque, a plaque is a buildup of a little
4:53 little
4:57 fibrous material inside the artery
4:59 and that doesn't cause a heart attack. A
5:02 heart attack is caused when one of those plaques
5:03 plaques
5:06 cracks. Now in this diagram for example,
5:08 it's showing that the plaque is closing
5:10 up the artery and this will cause
5:12 angina. That means a lack of blood
5:14 supply over here. So this patient may
5:16 complain of chest discomfort or he may
5:18 not complain of chest discomfort because
5:20 remember when you have a blockage like
5:23 this only 20% of the patients actually
5:25 get chest pain.
5:27 That means that you could have a
5:29 blockage like this in a patient and you
5:31 may not have any chest discomfort and
5:33 the only way you're going to pick that
5:35 up is by doing a stress test or a
5:38 coronary calcium score or some other
5:41 modality to see whether he has blockage
5:44 or not. A heart attack is when a minor
5:46 blockage which is not causing much flow
5:49 disturbance suddenly cracks
5:51 and what happens to the the plaque that
5:52 come that cracks off
5:55 and when it cracks open the blood that's
5:57 going past it sees the crack and wants
6:00 to repair it and forms a blood clot on
6:03 it. So the final thing that shuts down
6:06 that artery is a blood clot. It's not
6:08 the plaque that shuts it off, it's a
6:11 blood clot. So a heart attack is caused
6:14 by a blood clot at the sight of a
6:18 rupture and what causes rupture is inflammation.
6:19 inflammation.
6:22 Okay. So when people say that heart
6:25 disease is the number one killer of
6:26 people generally that are that are
6:28 diseased. Is it that process that's
6:30 causing it alone or is there a multitude
6:33 of processes that lead to heart disease?
6:35 There's a multitude of things that lead
6:37 to heart disease. Look,
6:39 that was a heart attack. That's right.
6:43 So, one, you're making plaque. You
6:45 shouldn't be making plaque.
6:48 A plaque is an inflammatory
6:51 area inside your arteries. It's reacting
6:53 to something. And I can go into the
6:55 nuance of what's actually what's the
6:57 body reacting to? What's inflammation?
6:58 Inflammation is reacting to something.
7:00 What is that something? And I think it's
7:01 important for your audience and
7:03 everybody to know what that something
7:06 is. Now, you've got this pimple. Think
7:08 of it as a pimple on the inside of the artery.
7:09 artery.
7:11 Now, one day when there's enough
7:14 inflammation in your body systemically,
7:16 that pimple is going to crack open just
7:19 like a pimple on the skin breaks open.
7:21 Now, that artery has this little pimple
7:23 on the inside and it cracks open and a
7:25 blood clot forms on it and that's a
7:28 heart attack. So, you don't want plaque
7:30 formation. You don't want plaque
7:33 instability which is rupture of that
7:35 plaque and you don't want a blood clot
7:37 to form on it. There are factors that
7:40 affect each of these processes and all
7:42 three processes are going wrong today
7:44 and that's why we have so much plaque
7:48 formation and instability of the plaque
7:49 which then leads of course to heart
7:53 attacks and our blood in general. We
7:56 have more coagulation issues today than
7:58 we ever did before. Which means our
8:00 blood because of inflammation is more
8:03 ready to clot more easily. So we having
8:05 the perfect triad.
8:08 It's the perfect storm. That's why we're
8:09 seeing an epidemic of this. And that's
8:11 why we're not making a dent in it. Yes,
8:13 you can have a plaque rupture and have a
8:15 heart attack and we put a stent in you.
8:18 Okay, that's fine. But what did we do to
8:21 look at the underlying reason as to why
8:22 you formed that plaque and why did you
8:25 rupture it and why was your blood so
8:27 clotty in the first place that made you
8:30 actually seal off the artery?
8:33 Is this an increasing problem?
8:36 Yes, it's a it's a huge problem because
8:38 those factors have been identified now
8:40 and that's what we go for in my
8:43 practice. We look for the factors. Why
8:45 did you make this plaque?
8:47 Why is there so much inflammation in
8:50 your body? Inflammation is a reaction to
8:52 something foreign. What is that foreign
8:55 stuff? What is that abnormal physiology
8:56 in you that your body is reacting
8:58 against? Cuz that's what inflammation
9:01 is. And inflammation, of course,
9:02 whenever there's going to be
9:02 inflammation, there's going to be
9:04 increased tendency to make blood clots.
9:07 So, we go in, we chase all these things
9:10 in patients to find out. And that's the
9:12 real prevention.
9:16 that 28-year-old that came in um and you
9:19 treated him, was it was that a surgery
9:19 you did?
9:22 We call it a surgery, but it's not. It's
9:24 a it's a angoplasty. So, we go in from
9:26 the wrist. Yeah.
9:26 Yeah.
9:28 Or we go in from the groin with a
9:30 catheter and we go into the coronary artery.
9:31 artery.
9:33 A catheter being like a tube.
9:35 It's a hollow tube. And we put it into
9:36 the coronary artery. We shoot the dye
9:38 inside. We can see where the blockage
9:42 is. And then we thread a tiny wire into
9:44 that artery through the blockage and
9:47 then bring in a balloon and squash the
9:50 blood clot out of the way. Restore the circulation
9:52 circulation
9:54 and then bring in a stent. A stent is
9:57 like a spring that opens the artery up.
9:58 We leave the stent inside there. We
10:00 remove everything else and now you have
10:03 an open artery and you have blood flow
10:04 going down that artery.
10:06 And what had that 28-year-old done to
10:09 end up in your surgery? What were the
10:10 decisions that they had made that
10:11 brought them there?
10:14 So, he was number one, he was
10:15 overweight, but he wasn't just
10:17 overweight. The overweight was all in
10:20 his belly and he had a fatty liver and
10:23 he had what is known as visceral fat. He
10:26 was not a diabetic, but he was a pre-diabetic.
10:28 pre-diabetic.
10:31 That means he had a lot of insulin in
10:33 his body and his glucose levels were
10:35 good. So, if you went to his family
10:36 doctor, they would say, "Oh, you're not
10:38 a diabetic. your hemoglobin A1C which is
10:41 a blood test that you do for diabetes is
10:43 actually okay but actually that patient
10:47 had a very high insulin blood level. Now
10:51 insulin is a very aogenic molecule. It
10:54 causes smooth muscle proliferation.
10:55 Smooth muscle is in the walls of the
10:58 arteries. It causes vasoc constriction.
11:01 It makes your blood more clotty and it
11:03 causes inflammation.
11:05 So this patient was a pre-diabetic
11:07 and this is very important and this is
11:10 what caught my attention because when I
11:12 looked at my patients that were coming
11:14 in with heart attacks and hardening of
11:16 the arteries and when I tried to
11:19 identify why they doing this none of
11:21 them were diabetics
11:24 but then I did a glucose tolerance test
11:27 on them and what happened it was just by
11:29 chance that I happened to have these
11:30 tests in my office where you can measure
11:33 the blood glucose. So I had about 120 of
11:35 them. So I said, "Look, let's just do
11:37 some random testing on these patients
11:40 and I found that at least half of them
11:42 had no diabetes but their glucose
11:45 intolerance. That means that the sugars
11:47 went up but not enough to make them a
11:50 diabetic but it did go above the normal
11:52 range. We did not have insulin testing
11:55 at that time. About 10 years later, we
11:57 started testing insulin in the office.
12:00 And I bought a machine for this purpose
12:02 in my office to measure insulin levels
12:05 and life was never the same again. Why?
12:06 Why?
12:08 Because it's all about insulin when it
12:10 comes to the metabolic derangement. So I
12:12 found that these patients the sugars
12:16 were going up to 150, 160 after giving
12:17 them a glucose drink. Mhm.
12:18 Mhm.
12:21 So you say, okay, he's not a diabetic.
12:22 Look, the sugar didn't really go high,
12:25 but when you measured the insulin level,
12:27 the insulin was off the roof.
12:29 So, when you say this is an insulin
12:30 problem, can you explain this to me?
12:32 Like, I have no idea what glucose and
12:34 insulin are for for any of my listeners
12:36 out there that have a inexperienced
12:38 understanding of these these these terms
12:39 and what they what they do and mean.
12:42 It's crucial. Everybody needs to
12:44 understand the relationship between
12:47 sugar, which is glucose, and insulin.
12:50 When you consume sugar or glucose, the
12:53 body has to get rid of that glucose very
12:55 quickly from the bloodstream because
12:57 glucose actually is toxic inside the
12:59 bloodstream. Even though it is what the
13:03 body uses for energy in the bloodstream,
13:05 it glycates all the blood vessels and
13:08 the walls and the components in blood
13:10 and hemoglobin as well glycates it. That
13:12 means a glucose attaches itself to that
13:14 molecule. So now that molecule can't
13:17 work properly. That is why the higher
13:19 your blood glucose,
13:21 all your chemicals don't work well, your
13:24 enzymes don't work well, your hormones
13:26 don't work well, nothing works well, and
13:28 you age prematurely because you're
13:31 getting glycation. Glyca, a glycated
13:33 molecule can't work normally. It doesn't
13:35 function normally.
13:38 So when you consume the glucose, the
13:40 glucose has to come out of the
13:41 bloodstream. And how does the body do
13:44 it? It sends a message to the pancreas.
13:46 The pancreas says, "Aha, I'll make some
13:48 insulin." It pours insulin into the
13:50 bloodstream. Insulin comes into the
13:52 bloodstream, pushes the glucose into the
13:55 cells. And where does it pushing it?
13:57 Into the liver, into the muscles, into
14:00 every cell in the body. Insulin will
14:03 push glucose out. Now, how much insulin
14:06 is the question.
14:10 If I'm eating every 3 hours and I'm
14:12 consuming glucose or I'm consuming
14:14 starchy foods or I'm consuming
14:16 carbohydrates. Now watch where I'm going
14:17 with this because you're already
14:18 beginning to know where I'm going with
14:21 this. I'm consuming glucose and carbs
14:24 every 2 three hours. I'm stimulating my
14:26 pancreas. I'm stimulating my insulin. My
14:28 insulin goes up, it comes down. But
14:30 before it even gets a chance to come
14:33 down, it goes up again.
14:37 So the repeated consumption of and
14:39 frequent consumption of glucose is
14:40 causing my insulin to stay high because
14:43 insulin stays a little bit longer in the
14:44 bloodstream than the glucose. The
14:46 glucose will come down in about 2 to 3
14:48 hours but the insulin stays higher for
14:51 about 4 hours. Now what happens is that
14:53 you continue this lifestyle for a few years.
14:55 years.
14:58 Now the body because these are all
15:01 hormones will say well you know it's I'm
15:03 going to need to make more insulin now
15:05 you become insulin resistant. Any
15:07 hormone that stays in your body for a
15:10 long time the body becomes immune to it.
15:12 So the next time I eat the sugar I'm
15:14 going to have to make more insulin to
15:17 produce the same effect. That is called
15:21 insulin resistance. So now you got this
15:24 patient who's been eating carbs, sugar,
15:26 sugar,
15:28 processed foods. What does that mean?
15:30 Processed foods that means foods that
15:31 are quickly absorbed into the
15:34 bloodstream. Processed foods, these are
15:37 products without fiber. So the
15:40 absorption is very quick. So the poor
15:43 pancreas has to react just like that.
15:46 Produces a whole bunch of insulin. And
15:48 then the frequent eating and the
15:50 frequent consumption makes you insulin
15:52 resistance. So now you produce a whole
15:54 bunch of insulin in order to bring that
15:57 sugar level down. So then you say, okay,
15:58 well it doesn't matter because insul the
16:00 insulin is bringing the sugar level
16:02 down. So what's the harm done? Because
16:03 your A1C is still good. You're not a
16:06 diabetic. No, but it's that background
16:09 high insulin that is destroying your metabolism.
16:10 metabolism.
16:12 It's that high insulin level in the
16:16 background. So insulin pushes glucose
16:18 into the liver and you develop a fatty liver.
16:20 liver.
16:22 It pushes the calories into production
16:25 of new fats around your viscera. The
16:28 viscera means in your belly around your
16:31 pancreas you get visceral fat. Now this
16:34 fat is produced from glucose. It's a
16:36 different kind of fat. Look if I gave
16:38 you a high calorie diet right now of all
16:39 sorts of foods you put on weight
16:42 everywhere. Okay?
16:44 But if I give you glucose, you put it on
16:47 mostly in your stomach and your stomach
16:49 will protrude and that's called visceral
16:51 fat. It's on the inside. You can't pinch
16:53 it. It's on the inside
16:54 around your organs
16:57 around the organs. This is very
17:00 detrimental fat. And that's the epidemic
17:02 that we have today. And that's a direct
17:04 result of eating, you know, starchy
17:06 glucose, carbohydrates, which creates
17:08 insulin, which creates this downstream
17:09 effect on
17:12 wrong foods and eating too frequently.
17:13 Too frequently. Okay?
17:14 Because remember, it's also the
17:15 frequency of eating because before that
17:17 insulin gets a chance to come down.
17:18 You're already popping yourself with
17:22 more food and hence you develop insulin
17:24 resistance. You get a very high insulin.
17:26 It takes this much insulin now just to
17:28 bring that sugar level down. And then
17:32 the one day when you cannot control that
17:35 sugar now that sugar will go out and now
17:37 your doctor will say you're a diabetic
17:39 but by that time you've already had 10
17:41 years of hyperinsulinemia.
17:43 So what happens if by the time you make
17:45 a diagnosis of diabetes to say well now
17:48 your sugar is really high it is high
17:50 because your body has not had the
17:52 ability to keep it down. Why? Because
17:54 even that high insulin could not keep
17:56 your sugar level down. You became a
17:58 diabetic. You lost that whole
18:00 opportunity of prevention. It's those 10
18:02 years. Look, by the time you're a
18:04 diabetic and you come and see Dr. Jay in
18:06 his cardiac clinic, you already have
18:08 coronary artery disease. Like that
18:11 28-year-old, that 28-year-old didn't
18:12 have diabetes. He already has coronary
18:15 artery disease. By the time you are
18:17 diagnosed as having diabetes, you
18:19 already have coronary artery disease. We
18:21 have a great opportunity here to
18:24 actually start screening these patients
18:27 with insulin levels very early on. But
18:29 most doctors don't have the ability or
18:30 knowledge to do the insulin level
18:33 testing, but it should be done.
18:36 In someone like me, I'm 33 years old
18:41 now. When does the damage begin?
18:44 It starts right now. It starts right
18:47 now. As soon as you around 30, you're
18:49 already starting to have trouble. You
18:51 have to But you know, this is what I do.
18:53 I look at that patient walking into my
18:56 room and if I see that he's got a belly
18:59 sticking out, I already know he's
19:01 probably got insulin resistance because
19:04 all the fat is in here. Because the fat
19:07 that's in the stomach, sideways, he
19:09 looks terrible. From the back, he looks
19:12 great. His waist is increased.
19:14 Mhm. He doesn't have all the fat
19:16 everywhere else in his body. That's the
19:18 phenotype of somebody who has hyperinsulinemia.
19:20 hyperinsulinemia.
19:22 That same person goes on a cruise, he'll
19:24 come back 5 to 10 pounds more because
19:27 he's got so much insulin in his body.
19:29 Insulin is a storage molecule. Puts
19:31 everything away. And it's very hard for
19:32 him to lose weight.
19:35 Why is it harder? you the only thing
19:36 that will make you lose that fat very
19:38 quickly is to change your diet of course
19:41 but you have to do fasting because
19:42 fasting brings your insulin level. See
19:45 this is where fasting comes in. So what
19:45 does fasting do?
19:47 Do you mean fasting or a calorie
19:48 deficit? Is it or is it the same thing
19:49 in your view?
19:51 No, they're not the same thing. They are
19:54 not the same thing. You see when you
19:57 don't eat your insulin levels come down
19:58 because you not stimulating your
20:01 pancreas anymore. So you want to bring
20:03 your insulin levels down. the best thing
20:06 you can do in the world is to do fasting
20:08 because there's no look if I just simply
20:12 cut down on my calories
20:14 then there's a different physiology
20:16 that's going to take place in the body
20:19 and when you fast there's a totally
20:21 different physiology when you cut down
20:24 on calories the body senses that this
20:26 caloric deficit your metabolic rate
20:30 changes actually slows down and the body
20:31 will start breaking down everything muscles
20:32 muscles
20:35 included. So you lose fat and you also
20:37 lose muscles. On the other hand, when
20:39 you're fasting, it's a different physiology.
20:41 physiology.
20:43 Fasting is I've put on fat. Now I'm
20:45 going to take it out of the bank. Now
20:47 the bank is going to be available for me
20:50 to pull out my calories and use it now.
20:52 And you start burning the fat. So in the
20:54 first 12 hours of a fast, you take out
20:56 all the glucose in the form of glycogen
20:59 from your muscles and your liver. After
21:02 12 hours, you start pulling the fat out.
21:04 And the first place the fat comes out of
21:07 is going to be visceral fat. That is why
21:09 fasting benefits you so much because it
21:11 gets rid of that worst fat. The fat that
21:14 is very inflammatory. You see, if I did
21:16 a biopsy of your visceral fat versus a
21:18 biopsy of let's say a fat form on your buttock,
21:20 buttock,
21:22 two different types of fat. One is full
21:25 of inflammatory molecules. The other one
21:27 is not full of inflammatory molecules.
21:30 One is producing interlucan 6 and tumor
21:33 necrosis factor and this other fat is
21:35 not. These are two different fat stoages.
21:37 stoages.
21:39 Visceral fat is very toxic. It's very
21:42 inflammatory. And that is why patients
21:44 who have visceral fat make all these
21:47 molecules. So when I do the blood test I
21:48 see that oh you've got so much
21:50 inflammation. You've got interlucan 6 is
21:52 high tumor necrosis factor is high. Your
21:55 CRP is running high. And one of the
21:58 reasons for this is not just leaky gut
22:00 and other things that I look at, but in
22:02 your case is also because you have a lot
22:06 of ectopic fat. Ectopic fat. Ectopic fat
22:09 is now we realizing is not just only
22:10 around the liver and around your
22:13 pancreas, it's also around your heart.
22:15 So when we look at the coronary arteries
22:18 and we see all those fat around the
22:19 coronary arteries, you had a very nice
22:22 diagram right there and you can see that
22:26 around each artery
22:29 there's that yellowess that yellow is
22:34 fat. There's fat around that and it's
22:37 plaque forming. It's plaque forming. It
22:40 stimulates plaque. It's inflammatory.
22:43 And now we can do CT scans that will
22:46 actually detect how much inflammation is
22:49 in the fat around the arteries as well.
22:52 Ectopic fat is in the around the
22:54 coronary arteries. It's in your liver
22:56 and in your pancreas and it is very inflammatory.
22:57 inflammatory.
23:00 On this point of fasting, so if you have
23:01 someone come to you and that, you know,
23:03 they have that physique where there's a
23:04 bit more of that protruding belly fat,
23:07 you said that fasting is a much better
23:08 approach than just sort of a calorie
23:10 restriction. was looking at some studies
23:12 that said research shows that calorie
23:14 deficits of any kind can reduce fiscal
23:16 fat, but fasting will give it an edge
23:18 because of the insulin sensitivity stuff
23:20 that you talked about as well. What kind
23:22 of fast should one be doing? Because
23:24 there's so many different names for
23:25 these fasts. People do these 40-day
23:27 water fasts and they do intermittent fasting.
23:28 fasting.
23:30 It's a great question. So, there are
23:32 many, many different types of fasting
23:36 depending on your goal on what you want
23:38 for that particular patient. So if a
23:41 patient is just simply looking to reduce
23:45 his visceral fat then I start with 1212
23:48 which means 12 hours you don't eat
23:50 anything. You just drink liquids with no
23:53 calories in it and then 12 hours is your
23:56 feeding period. We start with that and
23:58 we do that for about 2 to 3 weeks. Then
24:01 we quickly move to 186.
24:05 186 means 6 hours you get to eat. 18
24:09 hours. It's only
24:12 water, black tea, black coffee, green
24:16 tea, no calories. 186.
24:19 186. And then that's one type of
24:21 fasting. Now, if a patient is very
24:24 overweight, a patient has diabetes and
24:26 your goal is to reverse the diabetes,
24:29 the patient needs to lose 60 pounds,
24:31 then those patients have a special type
24:35 of need. For them, I will take them to a
24:40 48 hour fast once a week. Sometimes I'll
24:43 go to a three-day water fast every 9 days.
24:45 days.
24:48 Every 9 days, you will do OMAD. OMAD
24:50 means one meal a day only, every day for
24:53 9 days. And then you're going to give me
24:55 a three-day water fast.
24:58 Is the same advice applicable to women?
24:59 because obviously they're contending
25:02 with a variety of hormone fluctuations
25:05 and estrogen and I know that the the
25:06 female body responds differently to
25:10 these kinds of stresses like fasts.
25:11 I've been asked that question so many
25:14 times from patients as well. Most of the
25:16 women can actually handle it. The only
25:18 women that cannot are those who are
25:20 trying to become pregnant or or they
25:24 have um or they're already pregnant. I I
25:26 I think that women are not that
25:27 different when it comes to the fasting
25:30 programs. So, in my experience, I've
25:32 been able to get women to fast. I just
25:34 finished a fast on one lady just now for
25:40 72 days. 72 days. She was terribly
25:42 overweight. She had diabetes. She had
25:45 hypertension. She had hyper lipidmia.
25:48 She was having hip replacements, knee
25:50 replacements, joint problems. She had
25:53 skin problems. And we fasted her for 72 days.
25:54 days.
25:56 What did she have in those 72 days in
25:58 terms of drinks, electrolytes, coffee?
25:58 What was she?
26:00 Great question. So she would have black
26:04 tea, black coffee, water, and in the
26:06 water once a day I tell her to put some
26:09 electrolytes in there. So there's a
26:11 electrolyte called element or sometimes
26:12 I just tell them to go buy some Celtic
26:15 salt and put half a teaspoon in there
26:17 and once a day you take that. If you get
26:20 cravings and you feel really really
26:24 hungry, take some MCT oil, a teaspoon,
26:26 and put it in your water and you can
26:27 drink that as well. I
26:29 think it's worth saying that probably
26:31 you shouldn't try this at home, ladies
26:32 and gentlemen, because obviously medical
26:34 supervision is critical here. But in the
26:36 case of that lady, what was the before
26:40 and after of that 72-day fast? So,
26:43 diabetes gone, blood pressure normalized,
26:45 normalized,
26:47 weight loss, tremendous weight loss. I
26:50 mean, I think she lost about 55 60 lbs. Wow.
26:50 Wow.
26:53 Okay. And not only did she lose all that
26:54 weight, yes, all that weight from her
26:58 belly was gone, but even her face under
27:00 the arms. So, when you lose weight in a
27:02 fasting program, it's very different
27:05 from losing weight when you are
27:07 restricting calories. you actually
27:12 retract your your skin. So you get real
27:15 changes in your entire body. These
27:17 patient I had one patient that fasted
27:22 for 183 days under supervision. Okay. So
27:26 he went from 400 lb to 210 lb. And when
27:27 he walked into the office, you would not
27:29 recognize that he's just lost all this
27:33 weight because he did not look like skin
27:36 on top of bones and then having to have
27:38 surgery to remove all that excess skin.
27:40 Fasting is a totally different physiology.
27:42 physiology.
27:46 In fasting, you you you you the body is
27:47 doing a whole lot of things that are
27:49 very different. And we can go into the
27:51 physiology of fasting because that's
27:53 fascinating. It's a physiology that has
27:56 not been used by us. We've lost it. So
27:58 one of the things about modern living,
28:01 modern living, we have lost this
28:03 physiology of fasting. First of all, why
28:06 do we still have that physiology in us
28:07 is because it's supposed to serve a
28:09 purpose because we're supposed to be
28:12 fasting and feasting, but now we're only
28:14 feasting all the time and we're not fasting.
28:15 fasting.
28:18 In order to go back to normal, you
28:20 should be fasting. Fasting is supposed
28:23 to be a normal part of your existence.
28:26 That's the way you were designed. Not
28:28 this modern industrialized living that
28:31 we've been doing in the last fraction of
28:34 a millisecond in the total existence of
28:37 the human race. We've changed our
28:40 lifestyle so much. Modern man has. But
28:42 our genetics and our physiology has
28:46 lagged behind. We were supposed to use
28:48 fasting and feasting as part of our
28:50 normal program, as our normal
28:52 physiology. That's why we still have it.
28:55 That is why after 12 hours you start
28:58 making some more ketones in your body
29:00 and the ketones come from fat. You're
29:02 moving that fat out of storage.
29:03 What is a ketone?
29:06 Ah ketones. So ketones are a energy
29:09 molecule produced by the liver. How does
29:11 it make it? It makes it from fat. So the
29:13 fat gets liberalized. And by the way the
29:15 fats only start moving when your insulin
29:17 levels are down. So because of fasting
29:19 your insulin levels are really low now.
29:21 Okay? Because you've been fasting right?
29:24 So now the fats start dissolving. So you
29:26 get free fatty acids. The free fatty
29:28 acids float into the bloodstream. Free
29:31 fatty acids are fat products. They float
29:33 into the bloodstream. They go to your
29:35 liver. Your liver converts those into
29:37 ketones. Now ketones are an energy
29:39 source of the body, an alternative
29:43 source to glucose. So in general terms,
29:44 you are either going to be running on
29:48 glucose and glucose metabolism or you're
29:51 running on ketones. Okay? And so ketones
29:54 basically show up in the when glucose
29:55 isn't around.
29:57 That's absolutely right. So ketones and
29:59 ketones are actually a cleaner fuel for
30:02 the body. And in terms of producing
30:04 reactive oxygen species in the
30:07 metabolism, the way your mitochondria
30:09 work, you actually produce less reactive
30:12 oxygen species, which is damaging to
30:14 your physiology when you're in ketones.
30:17 And ketones are signaling molecules that
30:19 also change your physiology in a number
30:22 of ways. Number one, it causes the
30:24 production of brain derived neurotropic
30:28 factor that occurs under ketogenesis.
30:30 Brain derived neurotropic factor which
30:33 means that you become smarter, you're
30:35 growing new cells, your reflexes are
30:37 better, your visual acuity is better.
30:40 Why? Because nature wants you to become
30:41 a better specimen so you can go out and
30:43 get your next kill. So, why don't we all
30:45 just stay in a state of ketosis then if
30:47 it's so miraculous?
30:50 Well, that's a great question. You don't
30:52 want to be in ketosis all the time
30:53 because that's not what ketosis what
30:56 what our physiology was made for because
30:57 then you'll just be burning fats,
30:59 burning fats, burning fats all the time.
31:01 And that's not a good state to be in
31:04 constantly either. So, you want to be
31:06 able to do both. You're supposed to get
31:09 your current account and your deposit
31:11 account. You're supposed to work with
31:12 both of them. That's the normal
31:15 physiology. So not only brain derived
31:17 neurotropic factor increases but also
31:21 stem cells. Stem cells are amazing.
31:25 And in my patients of course I see that
31:27 what what is a stem cell?
31:30 Stem cells. So stem cells are we all
31:32 have stem cells and we all still make
31:35 stem cells and they are produced by the
31:38 bone marrow. These are pur potent cells.
31:41 Cells made that will then go out and
31:44 become whatever they need to become. So
31:45 they can go out into your circulation,
31:47 become a muscle cell, they can become a
31:49 retinal cell, a skin cell. They can
31:52 transform into anything. So what happens
31:55 is that when you break your fast Mhm.
31:57 you get a surge of stem cells coming out
31:59 of your bone marrow. Some of them become
32:01 immunocytes. That is why we know that
32:04 fasting also boosts your immunity.
32:06 People who fast get less infections, get
32:09 less sore throats and coughs and colds
32:11 and the viruses that are going around.
32:14 The immunity is better. The stem cells
32:15 are not I'm particularly interested in
32:19 the stem cells because of a thing called
32:21 the progenitor cells, endothelial
32:24 progenitor cells. Progentor cells are
32:26 you see you're always hurting your blood
32:28 vessels the lining of the blood vessels
32:29 and the lining of your blood vessels
32:31 have to be constantly repaired and they
32:33 they are repaired by the progentor
32:36 cells. When you do intermittent fasting
32:39 and timerestricted feeding you will
32:42 produce more and this has been shown in
32:45 numerous studies more progenitor cells
32:46 and these progentor cells go and repair
32:50 your blood vessels. So I do what why am
32:51 I interested in this? Because I'm a
32:53 vascular doctor, I want my blood vessels
32:56 to constantly be repairing themselves
32:58 from the damage that we do in day-to-day
33:02 life. So stem cell mobilization, brain
33:04 derived neurotropic factor, growth
33:07 hormone, you make more growth hormone in fasting.
33:08 fasting.
33:10 So when should you exercise when you're
33:14 fasting? At the peak of your fast. So,
33:16 if I'm going to break my fast at 6:00
33:19 p.m., I tell patients to go out to the
33:22 gym at 4:00 in the afternoon.
33:23 But I said, "But doc, how can I do
33:26 that?" You will find that you will put
33:28 on more muscle and you'll retain more
33:30 muscle as well because you have higher
33:32 growth hormone. The fastest way and the
33:33 best way to actually increase your
33:35 growth hormone production is to do
33:37 intermittent fasting.
33:40 When I sat with um Dr. Stacy Sims, who
33:42 does a lot lot of um work on women's
33:45 physiology and women's performance. One
33:47 of the things she said to me is that if
33:50 a woman in particular
33:54 tries to do a workout fasted, the body
33:57 will break down the muscle in a woman
33:58 because she said a woman's body is much
34:01 more the hippamus. It's much more
34:04 sensitive to changes in a woman's
34:05 glucose levels because it's trying to
34:06 defend against, you know, pregnancy and,
34:08 you know, all these kinds of things. So
34:09 it shuts down the menstrual cycle if
34:11 there's not enough glucose in the blood
34:13 and and so she said for women in
34:14 particular you do you do want to have
34:16 eat something before you work out or it
34:19 will your body will take from the muscle
34:21 and it will definitely won't the body's
34:23 very sensitive. It won't want to to help
34:24 you build muscle if there's not enough
34:26 glucose energy in the body. She said for
34:27 men it's slightly different because
34:29 we're a little bit more robust and we
34:31 were meant to hunt. So if we were
34:33 fasted, our body is, you know, designed
34:36 to kind of keep up its normal processes
34:39 to help us complete a hunt, per se. So I
34:40 guess I throw this question out to you
34:42 about about women exercise and fasting.
34:44 Yeah, I think that's a great
34:47 observation. So again is what kind of
34:49 exercise is the woman doing? You see,
34:50 now there's two kinds of basic
34:53 exercises. One is aerobic activity
34:56 and one is uh resistance exercises. And
34:57 you can also throw into that
35:01 highintensity interval training. Mhm. So
35:03 when it comes to the training that I'm
35:06 talking about in a fasting state, I like
35:09 resistance training. So in resistance
35:10 training, that glucose issue is not
35:11 going to become a problem. Now, if
35:13 you're going to put that female patient
35:14 on a treadmill and make her run for a
35:16 long period of time, let's say you're
35:17 going to make her run for 40 minutes on
35:19 the treadmill. I think that yes, you're
35:20 absolutely right. You are going to
35:22 deplete her glucose and you may cause
35:24 some problems and then the muscle will
35:26 start breaking down. But I do believe
35:27 from all the experience that I've had
35:29 with my patients that when I tell them
35:32 to do HIT, which is high intensity
35:35 interal training, all the women do just
35:37 as well as the men because all you're
35:39 doing is you're intensely working the
35:40 muscles and you're doing some resistance
35:42 exercise and then you're totally resting
35:44 as well. And I see fantastic results
35:45 with that.
35:48 In terms of ketosis,
35:50 do you cycle in and out of a ketogenic
35:52 diet? And just for anyone that doesn't
35:53 know what know, a ketogenic diet is a
35:54 diet that's very, very low in
35:57 carbohydrates. So less than 50 grams of
35:59 carbohydrates a day typically. Do you
36:01 cycle in and out of that diet?
36:04 Yes. Yes, you have to because I don't
36:07 want them to stay in a ketogenic state
36:10 all the time. So I do I do I do I tell
36:12 them, look, look, what is your goal
36:15 here? What is your goal? If your goal is
36:17 that, okay, I need to lose 70 lbs. Then
36:18 you're going to do this till you get to
36:19 your goal. Once you get to your goal,
36:20 then you're going to go back to
36:23 timerestricted feeding, which means 186.
36:25 So, you'll still make some ketones. And
36:28 then intermittently, you can still do a
36:32 36-hour fast. That means normal, healthy
36:34 people, you and me, we should still be
36:38 doing one 36-hour fast at least once a
36:40 month. So, we'll go to that. We'll go to
36:43 that. Fasting must still become part of
36:44 your pro, but you don't need to stay in
36:47 ketogenesis all the time. Remember also
36:50 that when you are in ketogenesis you are
36:54 you are also in autophagy.
36:57 How much autophagy can you do? That
36:59 means you recycling all your your partic
37:02 all your organels inside your cells.
37:04 What does that mean in simple terms for
37:05 someone that doesn't know the term autophagy?
37:06 autophagy?
37:12 So bottom line is the cell senses that
37:15 there is no new parts coming in here. So
37:17 it takes the redundant
37:21 organels inside the cells and breaks
37:24 them down, packages them up into these
37:27 little packets and exports them out of
37:30 the cells. So these cells, all your
37:34 cells in the body now are functioning at
37:36 a much more efficient level. So mitochondria
37:39 mitochondria
37:41 also have autophagy. So you're getting
37:43 new mitochondria. You're repairing your
37:44 mitochondria. And you know it's all
37:46 about mitochondria, right? Your energy
37:49 mitochondria are those organs organels
37:52 inside each cell. And we just think of
37:55 them as only an energy source that oh
37:58 yeah my mitochondria they make ATP.
38:00 That's not the whole story. Yes, they
38:04 produce ATP, but they also produce this
38:07 thing called reactive oxygen species
38:11 and thereby they influence
38:14 the metabolism of your of your cell as
38:16 well. They send signals they send
38:19 signals to your to your chromosomes so
38:21 you start producing new proteins. You
38:24 start producing new molecules. So your
38:27 mitochondria are very important. If you
38:28 have old mitochondria, you're going to
38:31 have fatigue, tiredness. But when you
38:34 get mphagy, one reason why you feel so
38:36 good after a fast because you have new
38:37 mitochondria, they are much more
38:40 efficient in producing ATP and less
38:42 reactive oxygen species. So you have
38:45 mphagy, recycling of your parts,
38:48 cellular function improves, toxins get
38:52 out of your cells as well, toxins.
38:55 Do you think there's a preferable or a
38:58 best exercise for heart health? Like if
38:59 you were going to say Stephen to make,
39:02 you know, my my I think my family has a
39:03 history of heart related issues. One of
39:06 my my uncle died from a heart attack. So
39:08 I'm very aware of
39:10 my own susceptibility to heart related
39:11 illnesses. I think there's other people
39:13 in my family um that have heart related
39:16 issues as well. Is there a particular
39:18 exercise that is best for the heart? Cuz
39:21 I'm guessing resistance training, i.e.
39:23 strength training might not be the the
39:26 number one recommendation for good heart
39:28 health. You mentioned HIT. I'm wondering
39:30 if that's
39:32 Yes. So, I'm just going to tell you
39:34 broadly speaking what I see in my
39:37 practice. I see that people who do
39:40 overly do aerobic activity. That means
39:43 they cycle 100 miles a day or they're
39:45 running on their treadmill for two hours
39:48 at a time every day. They're doing a lot
39:50 of lot of marathon training all the
39:52 time. They actually end up with more
39:53 inflammation in their body and they end
39:56 up with more coronary artery disease
39:59 than patients who do short sprints and
40:02 patients who do resistance exercises and
40:06 patients who do hits. So in terms of
40:08 cardiology, you look, you need some
40:10 aerobic training. Why? Because you want
40:12 to develop some endurance, right? You
40:13 don't want to be running behind the bus
40:16 and and get short of breath just in a
40:18 few hundred feet. So for some endurance,
40:21 you do aerobic activity. How much
40:23 aerobic activity which is running on the
40:25 treadmill for example or just sprinting?
40:29 Only about 15 to 20 minutes. That's it.
40:31 So I tell patients you want to run on
40:33 that treadmill, you want to bicycle 15
40:35 to 20 minutes and then you got to go
40:37 into resistance exercises. The
40:39 resistance exercises that I have seen
40:42 work best in my patients is simple
40:43 things. You don't have to go crazy. You
40:46 do flow exercises. You use your own body
40:49 weight as resistance. So you can do the
40:51 planks and leg lifts and all these other
40:54 exercise and you can do hit but they
40:56 have to be very specific. So you
40:59 exercise really hard for about 30
41:01 seconds to 45 seconds and then you
41:04 completely rest for another 30 to 45 seconds.
41:05 seconds. Okay?
41:06 Okay?
41:09 And that's allowing your body to clean
41:12 up the reactive oxygen species you just
41:13 created through that exercise. Because
41:17 your rate limiting step in cleaning up
41:19 the metabolic mess that you get when you
41:21 get too much exercise, it's a rate
41:22 limiting step. your glutathione
41:24 production, your superoxide dismutase
41:27 production. These are chemicals that
41:29 clean up the metabolic mess that you get
41:31 when you get too much activity. That's a
41:35 rate limiting step. So when you rest,
41:37 you exercise and you rest, you exercise,
41:40 you rest, you are going to get a cleaner physiology
41:41 physiology
41:43 with your patients. How many patients do
41:44 you think you've treated or worked with
41:46 in your career? Did
41:48 Did
41:50 you have to guess to the nearest thousand?
41:50 thousand?
41:53 It would have to be at least nothing
41:55 less than nothing less than quarter million.
41:56 million.
42:01 How do you deal with the heartbreak? And
42:03 that's no pun intended, but I'm saying
42:04 like how do you deal with the heartbreak
42:08 of watching people's lives
42:10 either them losing their life or their
42:12 their lives being changed permanently
42:14 forever or dealing with the families of
42:17 of people who lose their husband or wife
42:21 or or or son or daughter.
42:23 It it's very difficult especially when
42:25 we've been trying everything and still
42:28 things can go wrong. Based on that, I've
42:29 realized that sometimes you can do
42:32 everything to try to extend life, but
42:35 you cannot. Life is still finite. We
42:38 will pass. Yes, but we want better
42:41 health span and we want a longer
42:44 lifespan. I want to give the patients a
42:46 better quality of life also. I can't
42:49 make you live forever. I will see people
42:51 die. Sometimes they die in front of me
42:53 while I'm even holding their hands.
42:55 They'll pass away in the hospital right
42:59 there. But what I do want to do is to
43:02 give them a quality of life until then
43:04 that's going to change. And in all this education,
43:05 education,
43:08 it's all so that you can live an optimal
43:12 life and then pass away just like that.
43:14 Have you seen many people that made all
43:17 the right decisions? They were healthy.
43:20 They had a good diet. They were doing
43:24 exercise and they still end up passing
43:26 away on your hospital table because of
43:27 heart related illnesses.
43:28 Yes. Yes.
43:30 Give me one such example.
43:33 Typical example of fellow in the 60s.
43:35 His coronary calcium score was over
43:37 1,000. So you know that's a CT scan that
43:40 we do and it looks at calcium on the
43:42 arteries of the of the wall. So all this
43:45 plaque that you you you showed this
43:47 yellow stuff here, all that has calcium
43:50 in it. So the more calcium you have, the
43:52 more plaque you have. So he had a
43:55 calcium score of over a thousand. So sit
43:57 down and talk with him and say, "Okay,
43:59 so tell me why do you have all this
44:01 calcium? No diabetes, no high blood
44:04 pressure. Doc, I don't even drink. I
44:07 don't do anything. I don't smoke. I
44:09 exercise regularly. I go to the gym."
44:11 And then the question is, why do you
44:12 have all this coronary calcium in your
44:16 coronary arteries now? And it turned out
44:19 that ever since he was a kid, he had a
44:21 bad microbiome.
44:23 And in him, I found that that was the
44:25 reason why he got it. So he was shocked
44:28 when he left the office. He was totally
44:31 shocked because he thought that being
44:33 constipated and having a bowel movement
44:36 every three or four days
44:37 is just normal because it was always
44:41 like that for him until I dug into it
44:43 and found out that he's actually got a
44:46 lot of GI problems and that's what
44:49 probably caused him to have inflammation
44:51 and that's what's probably caused him to
44:53 have coronary artery disease. That's why
44:54 he's got all this coronary calcification.
44:56 calcification.
44:57 Yeah. So let's talk about the gut
44:59 microbiome then. You you mentioned
45:02 earlier that that's really pertinent in
45:04 cardiovascular issues. Can you explain
45:08 to me exactly why and how that happens?
45:10 The biggest difference between you and
45:13 your outer environment is actually your
45:17 gut, not outside your skin. It's
45:19 actually the gut. There's a 100 trillion
45:21 bacteria sitting in there and there's
45:24 probably 400 trillion viruses sitting in
45:26 your gut and they're not just hitching a
45:28 ride for you. They are there doing
45:30 something. There's a symbiotic
45:34 relationship between the gut and you.
45:36 They produce micronutrients which get
45:38 absorbed into your bloodstream. If the
45:40 right microbiome is not present, you are
45:42 being deprived of those micronutrients.
45:44 For example, short chain fatty acids
45:47 which are special kind of uh thing but
45:50 also they maintain the integrity of the
45:53 lining of the intestines. If the lining
45:56 of the intestines is breached then
45:59 bacteria wall products get into the
46:00 bloodstream and the first place they'll
46:04 get to is the liver. Hence you get a
46:07 fatty liver. There are basically three
46:08 reasons for fatty liver. Either you're
46:11 drinking too much alcohol or you get a
46:14 leaky gut or you have too much insulin
46:16 because insulin causes the conversion of
46:18 glucose into fat. Right? So you get a
46:19 fatty liver.
46:21 When you say leaky gut,
46:22 so that's a leaky gut.
46:24 Oh, okay. So leaky gut is when the
46:26 microbiome is dysfunctional.
46:28 The microbiome that is dysfunctional
46:31 causes the integrity of your intestinal
46:34 lining to be compromised.
46:34 Ah okay.
46:38 Yeah. So now molecules that are supposed
46:41 to stay in the gut, dead bacterial wall
46:44 products, they call lipopolyaccharides,
46:46 they're supposed to stay in the gut, but
46:48 now they get into the bloodstream, they
46:51 go up the portal vein, and up the portal
46:53 vein, they'll end up going straight to
46:55 the liver. And when they end up in the
46:57 liver, they cause that fatty liver. Now
46:59 you get this big fatty liver, which
47:02 causes a lot of inflammation. a fatty
47:05 liver. There's an epidemic of this going
47:08 on today. At least 25% of the population
47:09 now have a fatty liver.
47:12 And what is upstream from that? Is it is
47:14 it alcohol and all these processed foods
47:16 and things like that or
47:20 it's basically processed foods, sugar
47:22 and leaky gut and of course toxins.
47:24 Alcohol being the biggest toxin. Okay.
47:26 Okay.
47:28 This is the seat of metabolism. So, as a
47:30 cardiologist, I became interested in
47:32 this because I found that if you have a
47:35 fatty liver, you're going to be more
47:37 prone to inflammation
47:38 and you're going to be more prone to
47:41 coronary artery disease. When we fix the
47:43 fatty liver, we change the diet through
47:46 fatty by by changing your diet and now
47:48 you start eating real food and not
47:50 processed foods and products. when we
47:52 increase the fiber intake so that the
47:54 good bacteria start coming back into
47:57 your gut and when I give you certain
48:00 supplements to restore all your
48:02 nutrition in your body then all of a
48:05 sudden the progression of coronary
48:08 calcium flattens out. You see let's say
48:09 you came to me for a test and I did a
48:12 coronary calcium score and it's 500 and
48:16 two years from now it's 800. That means,
48:17 wait a second, something's not right
48:20 here, right? Your coronary calcium
48:23 should not be going up. Now, I can't get
48:24 rid of the coronary calcium because
48:25 that's plaque in the walls of your
48:28 arteries, but it shouldn't keep going up
48:30 because one day it's going to be 2,00.
48:33 So, how do I follow how well my
48:35 treatment for you is? Is I follow the
48:37 coronary calcium score.
48:38 And again, the coronary calcium score is
48:41 a marker of the amount of plaques,
48:44 correct? around the heart and in the
48:46 vessels of the blood.
48:46 Yeah. In the walls.
48:48 In the blood vessels. Okay. So, you want
48:49 low coronary calcium.
48:52 Like my calcium score is zero. You
48:54 shouldn't have any coronary calcium in
48:56 the walls of the arteries. So, we have
48:58 the yard sticks now.
48:59 We should probably we should probably
49:01 differentiate between calcium
49:03 supplements and the calcium that you're
49:04 talking about.
49:07 So, pertinent. Calcium supplements
49:08 actually increase the risk of
49:11 cardiovascular events. Calcium
49:13 supplements should not be taken. The
49:14 problem is not calcium supplements. The
49:18 problem is lack of D3 and lack of K2.
49:19 So, we shouldn't be taking calcium supplements.
49:20 supplements.
49:21 Absolutely not.
49:23 Absolutely. I stop all calcium
49:25 supplements on all my cardiac patients
49:27 and I tell them you should take vitamin
49:31 D3 so you'll absorb calcium better into
49:34 your gut and you take vitamin K2 because
49:37 vitamin K2 is going to make sure that
49:39 you don't get the calcium build up in
49:41 the wrong places in your vascule for example
49:41 example
49:43 just to give some context on the
49:44 research some some large observational
49:46 studies suggest that highdose calcium
49:49 supplements which is especially um above
49:52 1,000 milligs a day from pills may
49:53 increase the risk of heart attack and
49:55 stroke while others find no clear link. Yeah.
49:56 Yeah. Okay.
49:57 Okay.
50:00 Yeah. No, I look also they advise high
50:03 calcium supplements in women to prevent
50:05 osteoporosis for example, right? And
50:08 osteopenia. I live in Florida and all
50:09 these young women they come to me in
50:11 their 30s. They all have osteopenia. And
50:13 the question is that they got calcium
50:16 supplements put inside the milk in the
50:18 orange juice has calcium in it and
50:20 they're taking calcium supplements. Then
50:22 why would they and they have sunshine
50:24 and why would they have osteopenia? And
50:28 the reason is D3. You need D3 and K2.
50:30 That's what you need. It's a hormonal
50:33 thing. It's not the elemental thing.
50:35 So when people are on blood thinners, do
50:40 they typically have low vitamin K1?
50:42 If you're taking couadin, couadin is a
50:44 type of blood thinner. Yeah.
50:46 That lowers your your vitamin K1 levels
50:50 and it'll also lower K2. So couadin will
50:54 lower K2 as well. So you will get
50:55 increased coronary calcification and
50:57 that's been documented. So patients who
51:00 have who are taking warrin for example
51:02 or couadin have been shown to have
51:05 increased coronary calcification because
51:08 of the fact that the K2 levels come down
51:11 as well as the K1 levels both come down
51:13 both. So you need vitamin K2.
51:15 And you just do you supplement that or
51:17 do you get it from dietary? I supplement
51:19 it on all my patients because diet comes
51:22 from fermented foods and we don't eat
51:24 enough fermented foods to contain enough
51:27 vitamin K2. So one of the reasons why I
51:29 do like my supplements is because
51:33 today's food is not as rich as it was in
51:35 the past.
51:36 So we're not eating the variety of
51:38 foods, but also we're not getting enough
51:41 vitamin K2 in our regular diet. So, if
51:42 you want to give me advice then on
51:43 having a great gut so that I don't end
51:45 up with leaky gut which will cause all
51:47 of these downstream effects. What is um
51:49 what is the advice you'd give me to have
51:54 a perfect gut? Number one, lots of fiber
51:56 because the fiber is going to be eaten
51:58 by your bacteria and you're going to get
52:01 a wide variety of good bacteria eating
52:04 fiber. If you eat a lot of sugar, you'll
52:06 get bacteria too, but there are the bad
52:08 ones, the ones that you don't want. So
52:09 you're going to get a condition called
52:13 SIBO, for example. But you want to eat
52:16 lots of fiber. So because our diets
52:19 today don't give us enough fiber, I do
52:22 advise all my patients to take inulin
52:25 with FOS. It's called inulin with FOS.
52:27 It's a powder. It's a soluble fiber. And
52:29 I just tell them to take one scoop in
52:32 your water every day or put it in your
52:33 whatever you're going to have, whatever
52:36 liquid you're going to have. Number two,
52:38 fermented foods.
52:41 The fermented foods will give you not
52:44 only the bacteria themselves because,
52:47 for example, kefir has so many different
52:49 strains of good bacteria, but they also
52:51 have the postbiotics. What is a
52:54 postbiotic? A postbiotic, for example,
52:56 vitamin K2. It's a product that the
52:59 bacteria make.
53:01 Short chain fatty acids. That's a
53:03 product that the bacteria make. So, you
53:06 want bacteria. You want good bacteria in
53:09 your gut and they will stabilize the
53:11 ratio of all the other bacteria in your
53:14 gut. So the keystone species have to
53:16 come back into your gut.
53:19 I was reading some research around fiber
53:22 and it says that in the United States
53:25 women are 40% fiber deficient and men
53:29 are roughly 50% fiber deficient. And in
53:33 the UK we're about 40% fiber deficient.
53:35 I don't think people know that. Where do
53:37 I find fiber? What what do I need to be
53:40 eating to increase my fiber?
53:42 Fiber is found in anything that's nonprocessed
53:43 nonprocessed
53:46 because the very act of a processed food
53:48 is to take out the fiber so that you can
53:51 increase the shelf life of it. So any
53:52 processed food, anything that's
53:54 manufactured in a factory for example,
53:56 anything that already comes ready made,
53:58 they've taken the fiber right out of it.
54:01 So you got to eat real food. So you're
54:03 going to eat real vegetables and that to
54:05 a variety of vegetables. You see, you're
54:07 supposed to eat 30 to 40 different types
54:09 of vegetables a week and we don't tend
54:11 to do that. So you want fiber from a
54:14 variety of sources. So we got to eat a
54:15 little bit of all these different
54:17 different kind of vegetables including
54:20 spices. They all count as one vegetable.
54:21 So that's something I I stress to
54:23 everybody that you got to have variety.
54:24 You got to have all these different
54:26 fibers because they will foster the
54:27 growth of different types of bacterial
54:29 species in your gut.
54:31 What about lifestyle? recommendations
54:34 for improving my gut things like I don't
54:37 know stress sleep exercise anything
54:39 sleep absolutely important so the the
54:41 the next thing is actually actually
54:44 sleep and stress because lack of sleep
54:47 causes a change in your gut microbiome
54:51 you got to sleep 7 hours a night and
54:54 cutting down on sleep is going to affect
54:56 your entire physiology in your body and
54:58 your repair processes and your vagus
55:00 nerve and your delta sleep
55:03 But also your gut microbiome. One night
55:05 you'll get insulin resistance. One night
55:07 of bad sleep, you become insulin
55:09 resistant the next day.
55:12 If you're changing
55:14 if you're changing time zones very frequently,
55:16 frequently,
55:19 the liver dysfunctions
55:21 and the gut microbiome also dysfunctions.
55:23 dysfunctions.
55:26 So we got to be more cognizant of that.
55:29 That lifestyle is very important.
55:34 And we have to have plenty omega3 in the diet.
55:35 diet.
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56:38 You talk you talked earlier on about toxins.
56:39 toxins. Yeah.
56:40 Yeah.
56:42 What do you mean by toxins and how what
56:44 role is toxins playing in my physiology
56:46 and my heart health and my gut health?
56:47 Yeah. So,
56:49 and what is a toxin?
56:52 A toxin is a molecule
56:54 that should not be in your body. You
56:58 should not have been exposed to it. So,
57:01 let's take a pesticide or a herbicide.
57:03 These are chemicals
57:06 which man has made and not perhaps
57:10 adequately tested it in in um in
57:12 rigorous studies. And yet these
57:14 chemicals get into the body and we know
57:16 that many of them are estrogen receptor
57:19 disruptors and that causes inflammation
57:23 as well. So if you look at pesticides
57:25 and herbicides,
57:28 we look at plastics
57:31 forever chemicals for example PAS and
57:34 other chemical BPA.
57:36 These chemicals all disrupt our
57:39 metabolism in our body. And then of
57:42 course mold is a toxin. And lastly,
57:46 heavy metals are toxins.
57:49 When the gut is not working properly,
57:52 you're going to be more prone to
57:54 toxicity because the gut bacteria
57:56 normally grab a lot of those things and
57:58 take them out in your stools. So when we
58:00 look at toxicity, we look at gut health
58:03 and the liver health. The liver is
58:05 supposed to get rid of lot of toxins.
58:08 But in today's day and age, because of
58:10 the foods that we're eating, the
58:12 environment that we breathe, the
58:14 environment that we walk out into, we
58:16 getting too many toxins and this poor
58:19 organ here is getting overwhelmed and
58:22 cannot detoxify adequately. So when I do
58:25 a blood test, urine sample, and a stool
58:28 sample, I get a very good bird's eye
58:30 view of what's going on with their
58:33 ability to detoxify and which levels are
58:36 high. And I'm finding
58:38 that many of my patients who have gut
58:41 issues and liver issues are very, very
58:43 high with not only heavy metals, but
58:47 they also have a lot of pesticides,
58:51 herbicides, and mold in their body.
58:54 and mold. I'm finding a lot of mold and
58:58 mold and all these extraneous toxicities
59:00 cause inflammation. Inflammation is a
59:01 reaction to something that your body
59:02 shouldn't have.
59:04 Where is the mold coming from?
59:06 Mold is very interesting. I've only
59:08 started researching it in the last one year.
59:09 year. Basically,
59:12 Basically,
59:15 almost 70% of homes these days have some
59:17 form of mold toxicity in them, either
59:21 from previous water damage or exposure.
59:24 And mold is ubiquitous. It's it's just
59:26 about everywhere. And it can
59:28 reininoculate you every time you're in
59:31 that environment. and it causes your
59:34 innate immune system to start reacting
59:38 to it and causes lowgrade inflammation
59:41 in your body. And when I get rid of the
59:43 mold, all of a sudden I'm noticing that
59:44 the patients are not only feeling
59:47 better, but the rate of progression of
59:50 coronary artery disease flattens off. I
59:51 have this uh steam room in one of my
59:53 houses and uh I went in there the other
59:55 day and I smel I could smell it smelt a
59:58 bit funny and I recognized that smell
60:00 from when I was younger as being mold. So I like I googled it what what is that
60:03 So I like I googled it what what is that like earthy strong pungent smell in my
60:05 like earthy strong pungent smell in my steam room. I think what's happened is
60:08 steam room. I think what's happened is part of the like seat has come away from
60:10 part of the like seat has come away from the wall so it's not sealed anymore and
60:12 the wall so it's not sealed anymore and I think the moisture has got in there
60:14 I think the moisture has got in there with the water and I think it's moldy
60:16 with the water and I think it's moldy and chachi said to me when I was asking
60:18 and chachi said to me when I was asking chap said smell near the like cracks
60:21 chap said smell near the like cracks yes
60:22 yes and if you smell if it smells really
60:23 and if you smell if it smells really strong near the cracks then that's mold
60:25 strong near the cracks then that's mold and I smelled near the crack and it was
60:26 and I smelled near the crack and it was really really strong remember thinking
60:28 really really strong remember thinking oh like so I had I not known that would
60:31 oh like so I had I not known that would have been sitting in there 30 minutes a
60:33 have been sitting in there 30 minutes a day inhaling all of that strong mold
60:38 day inhaling all of that strong mold smell. And from doing this show, I've
60:40 smell. And from doing this show, I've I've learned that the downstream impacts
60:42 I've learned that the downstream impacts that can have on your body and your
60:44 that can have on your body and your cardiovascular health and everything in
60:45 cardiovascular health and everything in between is really, really bad. So, I I
60:47 between is really, really bad. So, I I had a team come over and just uh fix
60:49 had a team come over and just uh fix everything, repair it, and do a deep
60:51 everything, repair it, and do a deep clean. But I don't think people are very
60:52 clean. But I don't think people are very aware of how mold, which we kind of
60:55 aware of how mold, which we kind of think of as like, oh, the the the bread
60:57 think of as like, oh, the the the bread has gone green,
60:58 has gone green, whatever, can have a chronic downstream
61:01 whatever, can have a chronic downstream impact. And also this mold can come in
61:03 impact. And also this mold can come in and then become part of your microbiome.
61:06 and then become part of your microbiome. So it can actually come into your so
61:08 So it can actually come into your so let's say you get out of this place. But
61:10 let's say you get out of this place. But if the mold is still in your gut or if
61:12 if the mold is still in your gut or if it's in your sinuses for example and you
61:14 it's in your sinuses for example and you get chronic sinusitis you're
61:16 get chronic sinusitis you're reininoculating yourself over and over
61:18 reininoculating yourself over and over again with this mold. And this is an
61:21 again with this mold. And this is an area of great interest and a lot of
61:23 area of great interest and a lot of research needs to be done. It is real
61:26 research needs to be done. It is real and we've known about it for a long
61:27 and we've known about it for a long time. But the medical profession as a
61:29 time. But the medical profession as a general has not really taken this and
61:31 general has not really taken this and run with it, but it is very important
61:34 run with it, but it is very important and we certainly looking at mold
61:37 and we certainly looking at mold toxicity in our practice because we find
61:39 toxicity in our practice because we find that it is actually more prevalent than
61:40 that it is actually more prevalent than we ever ever imagined before and we know
61:43 we ever ever imagined before and we know the chemistry. We know that it causes a
61:46 the chemistry. We know that it causes a a systemic inflammatory reaction in the
61:48 a systemic inflammatory reaction in the body and it's reflected in the blood
61:50 body and it's reflected in the blood work. We see that the CRP is up. We see
61:52 work. We see that the CRP is up. We see that the compliment levels are high. we
61:54 that the compliment levels are high. we see that the certain ratios of certain
61:56 see that the certain ratios of certain inflammatory molecules are off. So we do
61:59 inflammatory molecules are off. So we do want to treat mold. Um we want to treat
62:01 want to treat mold. Um we want to treat sources of mold toxicity both in the gut
62:03 sources of mold toxicity both in the gut as well as in the sinuses. And yes,
62:07 as well as in the sinuses. And yes, people are surprised when I tell that
62:08 people are surprised when I tell that that affects the cardiovascular health.
62:11 that affects the cardiovascular health. When we talk about the clogging of
62:13 When we talk about the clogging of arteries, why is it that healthy people
62:16 arteries, why is it that healthy people still end up sometimes with these
62:18 still end up sometimes with these clogged arteries? I think it's because
62:20 clogged arteries? I think it's because they have sources of inflammation that
62:23 they have sources of inflammation that we have not identified. For example, we
62:25 we have not identified. For example, we say, "Oh, you have no cholesterol, you
62:28 say, "Oh, you have no cholesterol, you have no smoking, you have you're
62:30 have no smoking, you have you're exercising, you're not overweight, you
62:31 exercising, you're not overweight, you don't have diabetes." Well, then I don't
62:33 don't have diabetes." Well, then I don't know why you got all these odd. No,
62:35 know why you got all these odd. No, there's always if you dig deeper inside,
62:37 there's always if you dig deeper inside, there's always something. And we'll find
62:39 there's always something. And we'll find the source of the inflammation. There's
62:41 the source of the inflammation. There's always a reason why you get hardening of
62:43 always a reason why you get hardening of the arteries.
62:44 the arteries. What's in that list?
62:45 What's in that list? In that list is mold.
62:46 In that list is mold. Mold. Yeah.
62:48 Mold. Yeah. Licky gut. Yeah,
62:49 Licky gut. Yeah, huge food sensitivities. Oh, let me tell
62:52 huge food sensitivities. Oh, let me tell you about food sensitivities. So,
62:55 you about food sensitivities. So, another reason for the leaky gut is that
62:59 another reason for the leaky gut is that certain foods punch holes into your
63:02 certain foods punch holes into your intestines in a in a in a sense and
63:04 intestines in a in a in a sense and causes a leaky gut. And I've had at
63:06 causes a leaky gut. And I've had at least one patient in the last 6 weeks
63:09 least one patient in the last 6 weeks that I can remember who actually has
63:12 that I can remember who actually has celiac disease and he didn't even
63:14 celiac disease and he didn't even realize he had celiac disease because he
63:16 realize he had celiac disease because he had no constipation, diarrhea and he had
63:19 had no constipation, diarrhea and he had premature coronary artery disease and we
63:22 premature coronary artery disease and we identified that he had celiac disease.
63:24 identified that he had celiac disease. We cut out all weight from his diet and
63:26 We cut out all weight from his diet and that's going to stop this inflammatory
63:29 that's going to stop this inflammatory process from occurring. See, it's all
63:31 process from occurring. See, it's all about inflammation. We need to get rid
63:32 about inflammation. We need to get rid of inflammation in order to get rid of
63:34 of inflammation in order to get rid of coronary artery disease. Any source of
63:36 coronary artery disease. Any source of inflammation,
63:38 inflammation, get rid of the source. So if the
63:41 get rid of the source. So if the pesticide levels are high, herbicide
63:43 pesticide levels are high, herbicide levels are really high, get off that
63:46 levels are really high, get off that source. Maybe you maybe you're living on
63:48 source. Maybe you maybe you're living on a golf course and you smell those
63:50 a golf course and you smell those chemicals every day. Maybe you're using
63:51 chemicals every day. Maybe you're using it yourself in your yard. Maybe you have
63:54 it yourself in your yard. Maybe you have toxicity coming from mold as you
63:56 toxicity coming from mold as you mentioned. But we got to find the source
63:58 mentioned. But we got to find the source of it. So, a thorough evaluation of the
64:01 of it. So, a thorough evaluation of the gut microbiome, a thorough evaluation
64:06 gut microbiome, a thorough evaluation of toxins in your body, and a
64:08 of toxins in your body, and a nutritional status.
64:10 nutritional status. Your body is able to work and get rid of
64:14 Your body is able to work and get rid of this type of pathology if you have good
64:17 this type of pathology if you have good nutrition. We don't have good nutrition
64:20 nutrition. We don't have good nutrition today.
64:20 today. Let's talk about nutrition then. If I
64:22 Let's talk about nutrition then. If I wanted to end up on your surgical table,
64:25 wanted to end up on your surgical table, if I if that was my goal, what would you
64:28 if I if that was my goal, what would you recommend
64:30 recommend that I ate and yeah consumed?
64:34 that I ate and yeah consumed? You should eat nutrient
64:37 You should eat nutrient deficient
64:39 deficient processed foods every day.
64:43 processed foods every day. Give me some examples of of things.
64:45 Give me some examples of of things. Pringles and
64:47 Pringles and correct. So it'll be things made out of
64:50 correct. So it'll be things made out of wheat, refined wheat, biscuits, cookies,
64:56 wheat, refined wheat, biscuits, cookies, cakes, lots of bread. Basically,
65:01 cakes, lots of bread. Basically, fast foods,
65:03 fast foods, foods that come in a box, readymade
65:07 foods that come in a box, readymade food with labels,
65:10 food with labels, anything that comes in a packet that you
65:11 anything that comes in a packet that you open up,
65:13 open up, including some of these
65:16 including some of these that are high protein bars or some of
65:20 that are high protein bars or some of these because they have all sorts of
65:21 these because they have all sorts of other chemicals in them.
65:23 other chemicals in them. And I would have lots and lots of um
65:27 And I would have lots and lots of um artificial colorings in my food in all
65:30 artificial colorings in my food in all those processed foods. Um artificial
65:33 those processed foods. Um artificial sweeteners,
65:35 sweeteners, lots and lots of diet drinks and sugary
65:37 lots and lots of diet drinks and sugary drinks.
65:38 drinks. Orange juice.
65:39 Orange juice. Orange juice is is certainly on my list
65:42 Orange juice is is certainly on my list because that produces such a profound
65:44 because that produces such a profound increase in my glucose level and then my
65:46 increase in my glucose level and then my insulin spike that I'm going to get
65:47 insulin spike that I'm going to get afterwards will cause me to become very
65:49 afterwards will cause me to become very hungry 3 to four hours later. So yeah, I
65:53 hungry 3 to four hours later. So yeah, I would not eat real food
65:56 would not eat real food and you'll end up on my table.
66:00 and you'll end up on my table. See, alcohol, smoking,
66:01 See, alcohol, smoking, alcohol, no fiber because none of those
66:04 alcohol, no fiber because none of those foods will have fiber in them. None of
66:06 foods will have fiber in them. None of those processed foods have fiber in
66:08 those processed foods have fiber in them.
66:08 them. Your patients must come to you at times
66:11 Your patients must come to you at times and tell you what they're doing in terms
66:14 and tell you what they're doing in terms of what they're eating and lifestyle
66:16 of what they're eating and lifestyle choices. And there must be certain
66:17 choices. And there must be certain things which very people aren't aware
66:21 things which very people aren't aware aren't healthy. You actually mentioned
66:23 aren't healthy. You actually mentioned bread.
66:24 bread. Yeah.
66:25 Yeah. Is all bread bad?
66:27 Is all bread bad? No, not all bread is bad. Most bread is
66:30 No, not all bread is bad. Most bread is bad. I don't mind some sourdough because
66:32 bad. I don't mind some sourdough because it's fermented. So, it's going to get
66:34 it's fermented. So, it's going to get rid of some of those lectins that are
66:36 rid of some of those lectins that are already in the flour. So, those lectins
66:38 already in the flour. So, those lectins are gone out because they've been
66:39 are gone out because they've been fermented. So, it's probably a little
66:41 fermented. So, it's probably a little bit okay. But again, not large amounts
66:44 bit okay. But again, not large amounts of it. You know, a slice once or twice a
66:46 of it. You know, a slice once or twice a week is fine, but really it's a survival
66:49 week is fine, but really it's a survival food. Bread is a survival food. Just
66:51 food. Bread is a survival food. Just empty calories. That's all it's going to
66:52 empty calories. That's all it's going to do.
66:53 do. Is there like a worse bread? Is there
66:55 Is there like a worse bread? Is there like one bread which is
66:56 like one bread which is Yeah. Pure white bread. Simple white
66:58 Yeah. Pure white bread. Simple white bread. The simple light white bread.
67:00 bread. The simple light white bread. It's pure calories, 70 calories right
67:03 It's pure calories, 70 calories right there. You're talking about just eat
67:04 there. You're talking about just eat have 10 teaspoons of sugar instead in
67:07 have 10 teaspoons of sugar instead in one slice. I mean, it just doesn't make
67:09 one slice. I mean, it just doesn't make any sense whatsoever.
67:10 any sense whatsoever. White rice.
67:11 White rice. White rice. I white rice is a staple
67:15 White rice. I white rice is a staple food of many people and they cannot live
67:16 food of many people and they cannot live without it. So I run into this problem
67:18 without it. So I run into this problem all the time. So I tell them that this
67:20 all the time. So I tell them that this is what you're going to do. You're going
67:21 is what you're going to do. You're going to first and foremost soak your rice in
67:23 to first and foremost soak your rice in water and then discard the water after
67:26 water and then discard the water after an overnight soak because it contains
67:28 an overnight soak because it contains arsenic in it and other heavy metals.
67:31 arsenic in it and other heavy metals. You'll be surprised how much arsenic
67:32 You'll be surprised how much arsenic there's in rice these days. You see see
67:35 there's in rice these days. You see see the problem we have our sourcing of our
67:37 the problem we have our sourcing of our foods. I mean here as a cardiologist has
67:40 foods. I mean here as a cardiologist has to talk to his patients about how to
67:41 to talk to his patients about how to cook rice.
67:42 cook rice. What is arsenic for people that don't
67:44 What is arsenic for people that don't know?
67:45 know? It's a heavy metal that is found in the
67:48 It's a heavy metal that is found in the soil and the water is in contaminated
67:52 soil and the water is in contaminated soil and water and in these patty fields
67:54 soil and water and in these patty fields are now full of arsenic.
67:56 are now full of arsenic. It's a poison, isn't it? People I watch
67:57 It's a poison, isn't it? People I watch a lot of true crime. People kill each
67:59 a lot of true crime. People kill each other with arsenic.
68:00 other with arsenic. It's a very very strong poison and it'll
68:04 It's a very very strong poison and it'll slowly kill you. Is there anything else
68:06 slowly kill you. Is there anything else on that list of thing misconceptions?
68:09 on that list of thing misconceptions? So, so the rice has to be soaked. Then
68:11 So, so the rice has to be soaked. Then you get rid of the water. Then you cook
68:13 you get rid of the water. Then you cook it with lots of water. Throw away the
68:15 it with lots of water. Throw away the rice again. I mean the throw away the
68:17 rice again. I mean the throw away the water, not the rice. And then you cool
68:20 water, not the rice. And then you cool the rice in the refrigerator
68:23 the rice in the refrigerator and then you eat the rice the next day
68:25 and then you eat the rice the next day by reheating it. Now what you've done is
68:27 by reheating it. Now what you've done is you've created resistance starch.
68:30 you've created resistance starch. Now, what's going to happen is that that
68:31 Now, what's going to happen is that that rice is not going to have the same
68:34 rice is not going to have the same insulin effect in your body and weight
68:36 insulin effect in your body and weight gain effect and changes in your
68:38 gain effect and changes in your metabolism because it's now got
68:40 metabolism because it's now got resistant starch. Resistant starch means
68:42 resistant starch. Resistant starch means that those molecules have all bound to
68:44 that those molecules have all bound to each other. They don't they resist
68:46 each other. They don't they resist absorption and digestion until they get
68:48 absorption and digestion until they get to your colon. And then who's going to
68:50 to your colon. And then who's going to eat it? Your bacteria. So, you're
68:52 eat it? Your bacteria. So, you're basically feeding the bacteria with your
68:54 basically feeding the bacteria with your rice. So, if you're going to have rice,
68:57 rice. So, if you're going to have rice, this is the way you're going to do it.
68:58 this is the way you're going to do it. So,
68:59 So, is there anything else on that list that
69:02 is there anything else on that list that people should be thinking about that
69:04 people should be thinking about that they probably consider to be healthy
69:05 they probably consider to be healthy right now?
69:06 right now? Yeah. Yeah. Yeah. Yeah. Yeah. Yeah. So,
69:07 Yeah. Yeah. Yeah. Yeah. Yeah. Yeah. So, it's also what you do to your food.
69:10 it's also what you do to your food. Look, you take a nice piece of fish, for
69:12 Look, you take a nice piece of fish, for example, and if you burn it and you
69:16 example, and if you burn it and you blacken it really bad, you're creating
69:19 blacken it really bad, you're creating what is known as advanced glycation end
69:21 what is known as advanced glycation end products. You fry something, you're
69:24 products. You fry something, you're creating advanced glycation end
69:25 creating advanced glycation end products. So, let me define that for
69:27 products. So, let me define that for you. If nature does not have the ability
69:31 you. If nature does not have the ability on its own to take
69:35 on its own to take glucose, protein and fat and combine
69:38 glucose, protein and fat and combine them at very high temperature,
69:41 them at very high temperature, but you can do it in your air fryer
69:44 but you can do it in your air fryer and you can do it outside when you when
69:46 and you can do it outside when you when you're blackening your food and over
69:48 you're blackening your food and over blackening your food. It's called
69:49 blackening your food. It's called advanced glycation end products. So when
69:51 advanced glycation end products. So when you over burn your food, when you
69:54 you over burn your food, when you overcook your food, you're creating
69:55 overcook your food, you're creating these molecules. Now you're consuming
69:57 these molecules. Now you're consuming these molecules and they have been shown
69:59 these molecules and they have been shown to cause a radical increase in the
70:02 to cause a radical increase in the inflammation in your body because your
70:03 inflammation in your body because your body reacts to those products. There's
70:05 body reacts to those products. There's receptors to get rid of those they
70:07 receptors to get rid of those they called rages and they get rid of these
70:09 called rages and they get rid of these products but you overwhelm them and now
70:11 products but you overwhelm them and now you get inflammation.
70:13 you get inflammation. And is there any other watch out foods
70:15 And is there any other watch out foods that you know people think are healthy
70:18 that you know people think are healthy but maybe if if I was trying to have an
70:20 but maybe if if I was trying to have an optimally healthy heart I should avoid
70:21 optimally healthy heart I should avoid it.
70:22 it. Is this going to surprise you? excessive
70:25 Is this going to surprise you? excessive fruit
70:27 fruit because I'm not a very big follower of
70:30 because I'm not a very big follower of fruit. Fruit should be eaten only in
70:31 fruit. Fruit should be eaten only in season because it has too much fructose
70:33 season because it has too much fructose in it.
70:34 in it. Fructose being the sugars.
70:35 Fructose being the sugars. Fructose is sugar and fructose causes
70:38 Fructose is sugar and fructose causes fatty liver. So our overconumption of
70:41 fatty liver. So our overconumption of fruit is another factor that is
70:44 fruit is another factor that is contributing to coronary artery disease
70:46 contributing to coronary artery disease and diabetes and fatty liver. And we
70:49 and diabetes and fatty liver. And we think of fruit as being a healthy thing.
70:52 think of fruit as being a healthy thing. You see, but fruit should only be
70:54 You see, but fruit should only be consumed really in the fall and in
70:56 consumed really in the fall and in season and in small amounts because that
70:58 season and in small amounts because that high fructose level really causes major
71:02 high fructose level really causes major changes in your metabolism. So, I'm not
71:04 changes in your metabolism. So, I'm not too front of fruit. You can eat fruit in
71:06 too front of fruit. You can eat fruit in season, small amounts of it, but you
71:08 season, small amounts of it, but you know, people go crazy about fruit. I
71:11 know, people go crazy about fruit. I mean, I had a patient who was having
71:13 mean, I had a patient who was having mental problems as well as
71:15 mental problems as well as cardiovascular disease. And he that's
71:17 cardiovascular disease. And he that's all he lived on is fruit morning, lunch,
71:19 all he lived on is fruit morning, lunch, and dinner. And the moment we stopped
71:21 and dinner. And the moment we stopped that his health changed completely. He
71:23 that his health changed completely. He got so much better.
71:25 got so much better. So excess of fruit is also no good.
71:28 So excess of fruit is also no good. Excessively cooked foods, processed
71:30 Excessively cooked foods, processed foods, refined products. And another
71:32 foods, refined products. And another one, vegetable seed oils. So any food
71:36 one, vegetable seed oils. So any food that you prepare or you buy that
71:38 that you prepare or you buy that contains vegetable seed oils is a no no
71:42 contains vegetable seed oils is a no no because vegetable seed oils are a
71:44 because vegetable seed oils are a product. And if you look at all of them,
71:48 product. And if you look at all of them, they're exceedingly high in omega6. And
71:51 they're exceedingly high in omega6. And the ratio of omega 6 to omega3 is what
71:54 the ratio of omega 6 to omega3 is what the problem we have today.
71:57 the problem we have today. We have far too much omega6, very little
72:00 We have far too much omega6, very little omega3. So anything that contains
72:03 omega3. So anything that contains vegetable seed oils. So I always tell my
72:06 vegetable seed oils. So I always tell my patients, get rid of that vegetable seed
72:08 patients, get rid of that vegetable seed oil bottle from your home.
72:10 oil bottle from your home. So what oil should I use instead? The
72:12 So what oil should I use instead? The oil at home, if you're going to use any
72:14 oil at home, if you're going to use any oil, is going to be olive oil, extra
72:17 oil, is going to be olive oil, extra virgin olive oil for your for your
72:19 virgin olive oil for your for your salad. And if you're going to do high
72:21 salad. And if you're going to do high temperature, put a little pat of butter
72:24 temperature, put a little pat of butter or use a little bit of ghee or you can
72:27 or use a little bit of ghee or you can use a little bit of coconut oil. This is
72:30 use a little bit of coconut oil. This is a bit more of a different question, but
72:31 a bit more of a different question, but you said earlier on that when a patient
72:32 you said earlier on that when a patient walks into your office, you can kind of
72:34 walks into your office, you can kind of look at them and generally see if their
72:37 look at them and generally see if their cardiovascular health is intact. And
72:39 cardiovascular health is intact. And we're generalizing here, but if I'm sat
72:42 we're generalizing here, but if I'm sat at home right now and I'm trying to
72:44 at home right now and I'm trying to figure out if I'm healthy as it relates
72:47 figure out if I'm healthy as it relates to my cardiovascular health, what are
72:49 to my cardiovascular health, what are the symptoms of cardiovascular poor
72:53 the symptoms of cardiovascular poor health? Like what throughout the whole
72:55 health? Like what throughout the whole body? What what should I can I sort of
72:57 body? What what should I can I sort of obviously don't want anyone to self
72:58 obviously don't want anyone to self diagnose at home, but what kind of
73:00 diagnose at home, but what kind of symptoms should I be looking at when I
73:02 symptoms should I be looking at when I you when you look at me? Let's do this
73:03 you when you look at me? Let's do this on me to make it easier.
73:05 on me to make it easier. Yeah. So number one, look at your
73:06 Yeah. So number one, look at your weight.
73:07 weight. Okay. If you're overweight, that's a
73:10 Okay. If you're overweight, that's a that's a that's one strike against you
73:12 that's a that's one strike against you already. Number two, if you're
73:13 already. Number two, if you're overweight, where is it? It's mostly on
73:14 overweight, where is it? It's mostly on the belly.
73:15 the belly. I'm overweight according to the BMI.
73:19 I'm overweight according to the BMI. But again, you got to look at BMI, you
73:21 But again, you got to look at BMI, you know, is a little crude. It's not a very
73:24 know, is a little crude. It's not a very accurate way of actually looking at it
73:25 accurate way of actually looking at it because some people are very muscular
73:27 because some people are very muscular and the BMI of 28 is actually perfectly
73:29 and the BMI of 28 is actually perfectly okay for them. It's the constitution I
73:31 okay for them. It's the constitution I look at and it's visceral fat. Now, I
73:34 look at and it's visceral fat. Now, I can't measure your visceral fat by
73:36 can't measure your visceral fat by looking at you, but I can tell if your
73:37 looking at you, but I can tell if your belly is protruded. Now, in my office, I
73:40 belly is protruded. Now, in my office, I do visceral fat analysis.
73:41 do visceral fat analysis. So, what else? I've I've I don't have a
73:43 So, what else? I've I've I don't have a protruding belly per se. Um, what else
73:47 protruding belly per se. Um, what else should I be looking for to understand if
73:48 should I be looking for to understand if my cardiovascular health is intact? Is
73:51 my cardiovascular health is intact? Is there I know
73:52 there I know just by looking at you physically.
73:53 just by looking at you physically. Yeah.
73:54 Yeah. No, I that's it basically is your BMI
73:56 No, I that's it basically is your BMI and your overall weight and must not
73:58 and your overall weight and must not have a belly. Just don't have a belly.
74:00 have a belly. Just don't have a belly. If you have a belly, you have a problem.
74:03 If you have a belly, you have a problem. And what about my joints and stuff like
74:05 And what about my joints and stuff like that? Is that an indicator of
74:07 that? Is that an indicator of Yes, if you have joint now symptoms, if
74:09 Yes, if you have joint now symptoms, if you're having joint pains, look, if
74:12 you're having joint pains, look, if you're having joint pains, people think,
74:13 you're having joint pains, people think, "Oh, it's just degenerative joint
74:15 "Oh, it's just degenerative joint disease." In my experience, what I have
74:17 disease." In my experience, what I have seen, it's all inflammation. Because
74:20 seen, it's all inflammation. Because when I change the diet, when I fix the
74:22 when I change the diet, when I fix the gut issues, when I fix the inflammation
74:25 gut issues, when I fix the inflammation in the body, they all come back and say
74:27 in the body, they all come back and say the joints got better. All of them
74:29 the joints got better. All of them uniformly. Especially patients who have
74:31 uniformly. Especially patients who have autoimmune disease.
74:33 autoimmune disease. For example, many patients have
74:34 For example, many patients have rheumatoid and the moment we change
74:36 rheumatoid and the moment we change their diet, their lifestyle, the
74:39 their diet, their lifestyle, the frequencies of eating, introduce a
74:40 frequencies of eating, introduce a little bit of fasting. So, we do
74:42 little bit of fasting. So, we do everything right. Right. We've changed
74:43 everything right. Right. We've changed the gut microbiome. We've we've made the
74:46 the gut microbiome. We've we've made the liver better by by by giving them some
74:48 liver better by by by giving them some supplements. We do intermittent fasting,
74:50 supplements. We do intermittent fasting, we do some exercises, we give them some
74:53 we do some exercises, we give them some stress management, we improve their
74:55 stress management, we improve their sleep. All of a sudden the joint pains
74:58 sleep. All of a sudden the joint pains get better, rheumatoid gets better, skin
74:59 get better, rheumatoid gets better, skin diseases get better, psoriasis gets
75:01 diseases get better, psoriasis gets better because it's the fundamental
75:03 better because it's the fundamental underlying part is all inflammation.
75:06 underlying part is all inflammation. This is a random one, but I'm just
75:08 This is a random one, but I'm just connecting dots from different podcast
75:09 connecting dots from different podcast guests I've had on the show. What about
75:11 guests I've had on the show. What about bad breath?
75:13 bad breath? Bad breath. Bad breath is real.
75:16 Bad breath. Bad breath is real. But what is it? Is my oral microbiome
75:19 But what is it? Is my oral microbiome linked to my heart health?
75:20 linked to my heart health? Yes. There's a there's a microbiome that
75:23 Yes. There's a there's a microbiome that goes all the way from your nose, mouth,
75:25 goes all the way from your nose, mouth, all the way down to the anus. And each
75:27 all the way down to the anus. And each one is specific and can predict whether
75:30 one is specific and can predict whether you're going to get cardiovascular
75:31 you're going to get cardiovascular disease or not. There's unequivocal data
75:33 disease or not. There's unequivocal data to show that if you have bad teeth, bad
75:35 to show that if you have bad teeth, bad dental hygiene, bad bacteria in your
75:38 dental hygiene, bad bacteria in your mouth, you're going to get valvular
75:39 mouth, you're going to get valvular disease such as aotic stenosis,
75:41 disease such as aotic stenosis, premature calcification of your aotic
75:43 premature calcification of your aotic valve, and you're going to get coronary
75:45 valve, and you're going to get coronary calcification. That's been proven
75:47 calcification. That's been proven unequivocally. Patients who have chronic
75:49 unequivocally. Patients who have chronic sinusitis in your So you think that
75:51 sinusitis in your So you think that that's benign? If you have chronic
75:52 that's benign? If you have chronic sinusitis,
75:53 sinusitis, what's chronic sinusitis?
75:55 what's chronic sinusitis? Chronic sinusitis, infections inside
75:57 Chronic sinusitis, infections inside your maxural sinuses, frontal sinuses
75:59 your maxural sinuses, frontal sinuses constantly and you get these headaches
76:02 constantly and you get these headaches and and you get the cold and your block
76:04 and and you get the cold and your block nose and you constantly cog up inside
76:06 nose and you constantly cog up inside here. That's inflammation. And often
76:10 here. That's inflammation. And often times that's also linked to premature
76:13 times that's also linked to premature coronary artery disease and inflammation
76:14 coronary artery disease and inflammation in your body. Especially when it's
76:15 in your body. Especially when it's fungal. There's a condition called
76:17 fungal. There's a condition called fungal sinusitis. Again it comes down to
76:20 fungal sinusitis. Again it comes down to mold. And this mold it causes that
76:23 mold. And this mold it causes that lowgrade inflammation in your body. And
76:25 lowgrade inflammation in your body. And this has been linked to coronary artery
76:27 this has been linked to coronary artery disease. See what's happening is we
76:28 disease. See what's happening is we transforming the whole definition of the
76:31 transforming the whole definition of the causes of coronary artery disease. We
76:35 causes of coronary artery disease. We have been so myopic in our definition of
76:38 have been so myopic in our definition of what causes coronary artery disease
76:40 what causes coronary artery disease whereas everything your entire lif style
76:45 whereas everything your entire lif style mental physical eating everything will
76:48 mental physical eating everything will affect your coronary arteries everything
76:51 affect your coronary arteries everything everything
76:52 everything do you think much about
76:54 do you think much about over stimulation so I mean really mean
76:56 over stimulation so I mean really mean like too much caffeine does does too
76:59 like too much caffeine does does too much caffeine play a role and I say this
77:01 much caffeine play a role and I say this in part because I think there's been a
77:02 in part because I think there's been a few times where I've had like a pre-work
77:04 few times where I've had like a pre-work workout or too many glasses of coffee
77:07 workout or too many glasses of coffee and I've got like heart palpitations and
77:08 and I've got like heart palpitations and you almost like feel like you're dying.
77:10 you almost like feel like you're dying. Yes. Yes. Yes. Caffeine. Caffeine is
77:13 Yes. Yes. Yes. Caffeine. Caffeine is very similar to the adrenaline molecule.
77:15 very similar to the adrenaline molecule. Now, why would you want to have too much
77:17 Now, why would you want to have too much adrenaline in your body? Because then
77:19 adrenaline in your body? Because then you're living in a state of existential
77:21 you're living in a state of existential threat constantly and then what happens?
77:25 threat constantly and then what happens? You don't get the offset either. So, you
77:28 You don't get the offset either. So, you don't get the parasympathetic. You're
77:29 don't get the parasympathetic. You're only in sympathetic all the time.
77:31 only in sympathetic all the time. Fight or flight
77:32 Fight or flight all the time. So caffeine generates that
77:35 all the time. So caffeine generates that and the physiology it'll look caffeine
77:37 and the physiology it'll look caffeine is not as benign as we think it is. Now
77:40 is not as benign as we think it is. Now there's nothing wrong with having one or
77:41 there's nothing wrong with having one or two coffees a day. That's fine. Okay.
77:43 two coffees a day. That's fine. Okay. But I know people who drink about six
77:45 But I know people who drink about six glasses or six cups of coffee a day and
77:47 glasses or six cups of coffee a day and and I think that that's just that's
77:49 and I think that that's just that's toxicity. You see that's toxicity. Too
77:52 toxicity. You see that's toxicity. Too much caffeine. And it's a diuretic. And
77:55 much caffeine. And it's a diuretic. And a diuretic will cause intravascular
77:57 a diuretic will cause intravascular volume depletion and that also turns on
77:59 volume depletion and that also turns on your neuro hormonal activation. So you
78:01 your neuro hormonal activation. So you become more sympathetic. You see? So
78:04 become more sympathetic. You see? So it's a vicious cycle makes you
78:06 it's a vicious cycle makes you dehydrated.
78:07 dehydrated. I was reading about this earlier cuz I
78:08 I was reading about this earlier cuz I did have heart health palpitations one
78:09 did have heart health palpitations one day because I think I drank too much
78:10 day because I think I drank too much coffee. Um and what I what I see here in
78:14 coffee. Um and what I what I see here in the research says that moderate caffeine
78:16 the research says that moderate caffeine is safe and even heart protective for
78:17 is safe and even heart protective for most people. But excessive caffeine when
78:19 most people. But excessive caffeine when you get, you know, above 600 milligrams
78:21 you get, you know, above 600 milligrams a day
78:23 a day um can raise blood pressure, trigger
78:27 um can raise blood pressure, trigger arythiums,
78:28 arythiums, arrhythmias.
78:28 arrhythmias. Arhrihythmias and stress the heart. Yes.
78:30 Arhrihythmias and stress the heart. Yes. Now, I want to tell you why coffee is
78:32 Now, I want to tell you why coffee is good. It's not good because of the
78:34 good. It's not good because of the caffeine. And so, it's not good that,
78:35 caffeine. And so, it's not good that, oh, I'm getting that caffeine, so it's
78:37 oh, I'm getting that caffeine, so it's good for me. No, cuz believe it or not,
78:39 good for me. No, cuz believe it or not, coffee has a lot of soluble fiber in it.
78:42 coffee has a lot of soluble fiber in it. So, there is some soluble fiber in it
78:44 So, there is some soluble fiber in it and it has polyphenols.
78:46 and it has polyphenols. So, coffee has polyphenols just like how
78:49 So, coffee has polyphenols just like how cocoa has, you know, cocoa, cacao, 85%
78:52 cocoa has, you know, cocoa, cacao, 85% cacao, chocolate, that's fantastic. It's
78:53 cacao, chocolate, that's fantastic. It's got soluble fiber cuz who's going to
78:55 got soluble fiber cuz who's going to consume the soluble fiber? It's actually
78:57 consume the soluble fiber? It's actually the bacteria. So a coffee a day is
79:00 the bacteria. So a coffee a day is actually good for you for a different
79:01 actually good for you for a different reason. It's good because it's good for
79:03 reason. It's good because it's good for your bacteria because it's getting
79:05 your bacteria because it's getting soluble fiber and it's getting
79:06 soluble fiber and it's getting polyphenols and all those polyphenols
79:08 polyphenols and all those polyphenols are consumed by your bacteria.
79:10 are consumed by your bacteria. They they call cacao, don't they? The
79:12 They they call cacao, don't they? The something of the heart. My girlfriend's
79:14 something of the heart. My girlfriend's quite spiritual and in her practice they
79:17 quite spiritual and in her practice they call it like the heart medicine. Cacao.
79:19 call it like the heart medicine. Cacao. Cacao. It is because it has a lot of
79:22 Cacao. It is because it has a lot of antioxidants and it has a lot of soluble
79:24 antioxidants and it has a lot of soluble fiber. So wait a second. It's a soluble
79:27 fiber. So wait a second. It's a soluble fiber. Yes, because that's fostering the
79:29 fiber. Yes, because that's fostering the growth of the good microbiome. The good
79:31 growth of the good microbiome. The good microbiome is then going to produce
79:33 microbiome is then going to produce postbiotics and the postbiotics are
79:36 postbiotics and the postbiotics are going to give you all the benefit that
79:38 going to give you all the benefit that you're going to need. So, it's going to
79:39 you're going to need. So, it's going to cause less leaky gut, less inflammation
79:42 cause less leaky gut, less inflammation and your entic nervous system, your
79:44 and your entic nervous system, your vagus nerve, your vag nerve is going to
79:47 vagus nerve, your vag nerve is going to be protected because we're destroying
79:49 be protected because we're destroying our vag nerve right now with the leaky
79:51 our vag nerve right now with the leaky gut. When people get heart palpitations,
79:53 gut. When people get heart palpitations, I imagine a lot of them message you
79:55 I imagine a lot of them message you because it's quite scary, isn't it, when
79:56 because it's quite scary, isn't it, when you feel your heart beating,
79:59 you feel your heart beating, what is typically going on and when is
80:01 what is typically going on and when is it cause for concern if you've got heart
80:03 it cause for concern if you've got heart palpitations?
80:04 palpitations? Yeah. If you have underlying structural
80:06 Yeah. If you have underlying structural heart disease, let's say you have
80:08 heart disease, let's say you have blocked arteries, let's say you have a
80:10 blocked arteries, let's say you have a cardiopathy, a valvular disease, and
80:11 cardiopathy, a valvular disease, and you're having an arrhythmia, which is
80:13 you're having an arrhythmia, which is palpitations, that is definitely
80:14 palpitations, that is definitely life-threatening. They need to come
80:16 life-threatening. They need to come straight to the office and we need to
80:17 straight to the office and we need to monitor you and find out what's going
80:18 monitor you and find out what's going on. But let's say you're otherwise
80:20 on. But let's say you're otherwise perfectly healthy, young person,
80:22 perfectly healthy, young person, perfectly healthy, no heart disease
80:24 perfectly healthy, no heart disease whatsoever, and you're having
80:25 whatsoever, and you're having palpitations, then it's usually an
80:27 palpitations, then it's usually an imbalance of your sympathetic and
80:29 imbalance of your sympathetic and parasympathetic nervous system. You're
80:31 parasympathetic nervous system. You're too stressed.
80:32 too stressed. It's basically either too much
80:34 It's basically either too much sympathetic or not too much sympathetic,
80:37 sympathetic or not too much sympathetic, but you have too little parasympathetic.
80:39 but you have too little parasympathetic. And that is one thing that I want people
80:41 And that is one thing that I want people to realize that if you've knocked your
80:43 to realize that if you've knocked your vag nerve off because you have a leaky
80:46 vag nerve off because you have a leaky gut or because of your lifestyle and
80:49 gut or because of your lifestyle and your vagus nerve is not working very
80:51 your vagus nerve is not working very well, you will have this imbalance
80:53 well, you will have this imbalance because the imbalance is not with
80:54 because the imbalance is not with absolute levels. It's the relative
80:56 absolute levels. It's the relative levels of sympathetic versus
80:57 levels of sympathetic versus parasympathetic. So for example, I see a
80:59 parasympathetic. So for example, I see a lot of young women in their 30s and 20s
81:01 lot of young women in their 30s and 20s who come to me with tachicardia. They
81:03 who come to me with tachicardia. They have a rapid heartbeat all the time and
81:05 have a rapid heartbeat all the time and especially when they stand up, they get
81:06 especially when they stand up, they get a rapid heartbeat. This is called
81:08 a rapid heartbeat. This is called postural orthostatic tachicardia. And
81:11 postural orthostatic tachicardia. And when they come to me, I find out that
81:14 when they come to me, I find out that it's actually the gut. I fix their gut
81:16 it's actually the gut. I fix their gut and the tachicardia gets better because
81:18 and the tachicardia gets better because by fixing the gut, I'm fixing the vagus
81:21 by fixing the gut, I'm fixing the vagus nerve. By fixing the vagus nerve, the
81:24 nerve. By fixing the vagus nerve, the heart rates come down.
81:26 heart rates come down. I've built companies from scratch and
81:28 I've built companies from scratch and backed many more. And there's a blind
81:30 backed many more. And there's a blind spot that I keep seeing in early stage
81:32 spot that I keep seeing in early stage founders. They spend very little time
81:34 founders. They spend very little time thinking about HR. And it's not because
81:36 thinking about HR. And it's not because they're reckless or they don't care.
81:38 they're reckless or they don't care. It's because they're obsessed with
81:39 It's because they're obsessed with building their companies. And I can't
81:41 building their companies. And I can't fault them for that. At that stage,
81:42 fault them for that. At that stage, you're thinking about the product. How
81:44 you're thinking about the product. How to attract new customers, how to grow
81:45 to attract new customers, how to grow your team, really how to survive. And HR
81:48 your team, really how to survive. And HR slips down the list because it doesn't
81:49 slips down the list because it doesn't feel urgent. But sooner or later, it is.
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82:35 justworks.com. I've just invested millions into this
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82:41 It's a company called Ketone IQ. And the story is quite interesting. I started
82:43 story is quite interesting. I started talking about ketosis on this podcast
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83:39 And I'm so honored that once again, a company I own can sponsor my podcast.
83:42 company I own can sponsor my podcast. Let's talk about the V Vegas nerve then.
83:44 Let's talk about the V Vegas nerve then. Um before we do that, I had a little a
83:48 Um before we do that, I had a little a little incident actually caused by
83:50 little incident actually caused by Jamaima who's out there. Jamaima We were
83:53 Jamaima who's out there. Jamaima We were in this little fitness competition and
83:54 in this little fitness competition and Jamaima decided that she wanted to beat
83:56 Jamaima decided that she wanted to beat me and so she cycled 100 kilometers one
83:59 me and so she cycled 100 kilometers one day. She cycled for 4 hours which meant
84:01 day. She cycled for 4 hours which meant that she had more minutes in this
84:03 that she had more minutes in this fitness competition than I did. So my
84:05 fitness competition than I did. So my rebuttal was to do the same but more. So
84:08 rebuttal was to do the same but more. So the next day I cycled just over 100 km
84:11 the next day I cycled just over 100 km on my Pelaton at home. Funny enough, it
84:13 on my Pelaton at home. Funny enough, it took me like 4 hours to do it. And then
84:15 took me like 4 hours to do it. And then I came off the Pelaton. I was
84:16 I came off the Pelaton. I was celebrating whatever, you know, won this
84:19 celebrating whatever, you know, won this gold medal emoji as my reward that
84:21 gold medal emoji as my reward that month, but also I won heart palpitations
84:24 month, but also I won heart palpitations that stayed with me for a little while.
84:26 that stayed with me for a little while. And I was wondering like why that
84:27 And I was wondering like why that happened. So I did this big vigorous
84:29 happened. So I did this big vigorous exercise which was kind of outside of my
84:31 exercise which was kind of outside of my usual exercise regime and then for a a
84:34 usual exercise regime and then for a a couple of weeks I could like feel these
84:36 couple of weeks I could like feel these heart pulpitations
84:38 heart pulpitations and is that because of what you were
84:39 and is that because of what you were saying about my parasympathetic?
84:42 saying about my parasympathetic? What do you think happened there?
84:43 What do you think happened there? Yes. Yes. Yes. Yes. over exercise.
84:48 Yes. Yes. Yes. Yes. over exercise. This has been shown when you do what you
84:50 This has been shown when you do what you just did, you're diverting blood from
84:53 just did, you're diverting blood from your gut to your muscles because your
84:56 your gut to your muscles because your muscles at that point really need all
84:58 muscles at that point really need all that blood supply. And this has been
84:59 that blood supply. And this has been well documented. So what happens when
85:02 well documented. So what happens when you overly exercise like that? You're
85:04 you overly exercise like that? You're causing relative eskeeia to your gut.
85:07 causing relative eskeeia to your gut. Essia. Essia. What does eskeemia mean?
85:09 Essia. Essia. What does eskeemia mean? Lack of circulation to your gut. So your
85:11 Lack of circulation to your gut. So your poor gut at that point had lack of
85:14 poor gut at that point had lack of circulation relative. Not to the point
85:16 circulation relative. Not to the point where it's going to go gangrous. No,
85:17 where it's going to go gangrous. No, don't don't get me wrong. But it affects
85:20 don't don't get me wrong. But it affects your gut and in the gut is the entic
85:23 your gut and in the gut is the entic nervous system. The vagus nerve ends in
85:26 nervous system. The vagus nerve ends in your gut lining and the vagus nerve got
85:28 your gut lining and the vagus nerve got affected. So your vagus nerve didn't
85:30 affected. So your vagus nerve didn't work very well. When you get vagus nerve
85:32 work very well. When you get vagus nerve not working well, you get too much
85:34 not working well, you get too much sympathetic. You're going to get
85:34 sympathetic. You're going to get tachicardia. You're going to get
85:36 tachicardia. You're going to get arrhythmias, palpitations. So tell me
85:39 arrhythmias, palpitations. So tell me what the vagus nerve is and what I can
85:41 what the vagus nerve is and what I can do to get my vagus nerve in check. By
85:44 do to get my vagus nerve in check. By the way, Jamaima, it was totally worth
85:45 the way, Jamaima, it was totally worth it. But just tell me what I what I can
85:47 it. But just tell me what I what I can do to get my Vegas nerve in check and
85:49 do to get my Vegas nerve in check and like what role my Vegas nerve plays.
85:50 like what role my Vegas nerve plays. I've got this photo here of the Vegas
85:52 I've got this photo here of the Vegas nerve.
85:53 nerve. Yeah, you know, the Vegas nerve is
85:56 Yeah, you know, the Vegas nerve is fascinating.
85:57 fascinating. It is the largest nerve in the body
86:01 It is the largest nerve in the body and it it it basically is sending
86:05 and it it it basically is sending messages from the brain to the whole
86:07 messages from the brain to the whole body and receiving messages back to say
86:10 body and receiving messages back to say what is the state of affairs and the
86:12 what is the state of affairs and the largest distribution believe it or not
86:14 largest distribution believe it or not is all in your gut. Now that tells you
86:17 is all in your gut. Now that tells you something right there that
86:20 something right there that what is the brain most concerned about?
86:23 what is the brain most concerned about? It's most concerned about the most
86:26 It's most concerned about the most treacherous border in your body which is
86:28 treacherous border in your body which is your gut.
86:30 your gut. Your gut health is so important that the
86:34 Your gut health is so important that the body has dedicated a huge nerve called
86:37 body has dedicated a huge nerve called the vagus nerve just to take care of
86:40 the vagus nerve just to take care of your gut because your gut is that
86:43 your gut because your gut is that important. That is why by the way it
86:46 important. That is why by the way it also has endings on the heart in your
86:48 also has endings on the heart in your lungs in your face. So there's lots of
86:51 lungs in your face. So there's lots of endings of the vagus nerve
86:53 endings of the vagus nerve and what is it doing? Is it telling is
86:54 and what is it doing? Is it telling is it basically the communication channel
86:56 it basically the communication channel between all your gut, your heart, your
86:58 between all your gut, your heart, your brain
86:58 brain state of affairs? What is going on in
87:00 state of affairs? What is going on in the gut? What should I be doing? Back
87:02 the gut? What should I be doing? Back and forth traffic information going back
87:04 and forth traffic information going back and forth. So when you when you the body
87:09 and forth. So when you when you the body is supposed to be in a state of
87:11 is supposed to be in a state of sympathetic followed by parasympathetic.
87:13 sympathetic followed by parasympathetic. What does that mean?
87:14 What does that mean? You run away from the tiger. You hide
87:17 You run away from the tiger. You hide behind the rock. The tiger is gone now
87:20 behind the rock. The tiger is gone now and now you're supposed to be
87:21 and now you're supposed to be parasympathetic.
87:22 parasympathetic. So sympathetic is the fight.
87:24 So sympathetic is the fight. Parasympathetic is the the relaxation,
87:26 Parasympathetic is the the relaxation, rest, relaxation, repair. Guess what?
87:29 rest, relaxation, repair. Guess what? We're not getting enough repair these
87:31 We're not getting enough repair these days because we constantly in the fight
87:33 days because we constantly in the fight flight fight. We never give our chance
87:35 flight fight. We never give our chance to go into repair. That's what the
87:38 to go into repair. That's what the problem with the Vegas nerve that we
87:39 problem with the Vegas nerve that we have today is. So we don't get a chance.
87:42 have today is. So we don't get a chance. So that is why don't we do it? Well, one
87:45 So that is why don't we do it? Well, one is our lifestyle. Two, our gut is not
87:49 is our lifestyle. Two, our gut is not right because it has disade our vagus
87:52 right because it has disade our vagus nerve dysfunctional. When I fix the gut,
87:54 nerve dysfunctional. When I fix the gut, one of the things I notice is heart rate
87:57 one of the things I notice is heart rate variability gets better. What does that
87:59 variability gets better. What does that mean? Heart rate variability. That means
88:01 mean? Heart rate variability. That means your pulse with each breath in and each
88:05 your pulse with each breath in and each breath out, there's a little variation
88:07 breath out, there's a little variation in your heartbeat. H that's called heart
88:09 in your heartbeat. H that's called heart rate variability. Now, when you lost
88:12 rate variability. Now, when you lost your heart rate variability, it means
88:14 your heart rate variability, it means that your vagus nerve is not working
88:15 that your vagus nerve is not working very well. In all patients when I fix
88:17 very well. In all patients when I fix the gut the heart rate variability gets
88:19 the gut the heart rate variability gets better as well. When you fix the gut
88:22 better as well. When you fix the gut your vagus nerve will be able to work
88:24 your vagus nerve will be able to work more efficiently and you'll be able to
88:26 more efficiently and you'll be able to repair yourself after the sympathetic
88:29 repair yourself after the sympathetic drive. So you get the ying and then you
88:31 drive. So you get the ying and then you get the yang as well. And the nice thing
88:33 get the yang as well. And the nice thing about the vagus nerve that I find is
88:34 about the vagus nerve that I find is that you can repair it through the gut
88:36 that you can repair it through the gut but you can also hack it and you can
88:39 but you can also hack it and you can give it some nutritional supplements.
88:41 give it some nutritional supplements. The nutritional supplements are omega-3
88:44 The nutritional supplements are omega-3 which right now 50% of the population is
88:46 which right now 50% of the population is very low in omega-3 and you need DHA and
88:49 very low in omega-3 and you need DHA and omega-3 to to to make their vag nerve
88:52 omega-3 to to to make their vag nerve work optimally. You need to fix the gut
88:54 work optimally. You need to fix the gut and thirdly you can hack the vagus
88:57 and thirdly you can hack the vagus nerve. So how do you do that? You do the
88:59 nerve. So how do you do that? You do the breathing exercises. So when you do the
89:02 breathing exercises. So when you do the deep expiration, you stimulate all those
89:06 deep expiration, you stimulate all those parasympathetic nerve endings and your
89:09 parasympathetic nerve endings and your heart rate goes down, blood pressure
89:11 heart rate goes down, blood pressure goes down.
89:12 goes down. What kind of breathing exercises?
89:14 What kind of breathing exercises? So breathe in to the count of four,
89:17 So breathe in to the count of four, breathe out to the count of eight. Very
89:19 breathe out to the count of eight. Very simple. I tell that to all my patients.
89:21 simple. I tell that to all my patients. Breathe in to the count of four. Breathe
89:22 Breathe in to the count of four. Breathe out to the count of eight. It's a
89:24 out to the count of eight. It's a deliberately long expression. You do
89:26 deliberately long expression. You do that for about 10 minutes a day.
89:28 that for about 10 minutes a day. And when you do that, your vagus nerve
89:31 And when you do that, your vagus nerve is getting stimulated. And any nerve
89:33 is getting stimulated. And any nerve that is stimulated frequently enough
89:35 that is stimulated frequently enough will work more efficiently.
89:37 will work more efficiently. What about this eyeball thing that I've
89:38 What about this eyeball thing that I've heard you talk about where you
89:40 heard you talk about where you eyeball does affect the vagus nerve
89:42 eyeball does affect the vagus nerve function too. So when you look to the
89:44 function too. So when you look to the right, look to the left at the top, look
89:46 right, look to the left at the top, look down, look left, it stimulates the vagus
89:48 down, look left, it stimulates the vagus nerve. There's a nerve on the ear.
89:51 nerve. There's a nerve on the ear. There's also the vagus nerve that can be
89:52 There's also the vagus nerve that can be stimulated here in the neck that can be
89:55 stimulated here in the neck that can be stimulated. The facial nerve also uh has
89:58 stimulated. The facial nerve also uh has branches that go to the vagus nerve. So
90:01 branches that go to the vagus nerve. So there are a few hacks that people can
90:03 there are a few hacks that people can use. I even have a small contraption
90:05 use. I even have a small contraption that I give patients that wear around
90:07 that I give patients that wear around the neck and it it it releases little
90:10 the neck and it it it releases little signals onto the vagus nerve over here
90:12 signals onto the vagus nerve over here and I've placed it on patients and they
90:15 and I've placed it on patients and they in front of me and they'll say, "Oh my
90:18 in front of me and they'll say, "Oh my god, I really feel so relaxed and nice."
90:20 god, I really feel so relaxed and nice." And it's I was shocked when they did
90:23 And it's I was shocked when they did that. I heard you talk about massaging
90:25 that. I heard you talk about massaging the eyeball.
90:26 the eyeball. Yes. And cold water on the eyeballs. So,
90:30 Yes. And cold water on the eyeballs. So, I don't advise massaging too often, but
90:32 I don't advise massaging too often, but cold water and gentle massage on your
90:34 cold water and gentle massage on your eyeballs stimulates the vagus nerve. And
90:36 eyeballs stimulates the vagus nerve. And you can do it yourself. You'll notice
90:37 you can do it yourself. You'll notice that if you're on a monoton, you do
90:38 that if you're on a monoton, you do that, your pulse rate goes down. It's
90:40 that, your pulse rate goes down. It's the deep dive reflex that we have in all
90:44 the deep dive reflex that we have in all of us. We dive into water.
90:46 of us. We dive into water. What about putting an ice pack on the
90:48 What about putting an ice pack on the back of your neck?
90:49 back of your neck? I think that's fantastic. Back of the
90:51 I think that's fantastic. Back of the neck, but I prefer the front of the
90:52 neck, but I prefer the front of the neck. Okay.
90:53 neck. Okay. Because the front of the neck is closed.
90:55 Because the front of the neck is closed. The vagus nerve is very close to the
90:56 The vagus nerve is very close to the karate sheath over here. And when you
90:59 karate sheath over here. And when you put ice packs on it, the vagus nerve is
91:01 put ice packs on it, the vagus nerve is stimulated.
91:02 stimulated. Humming for 10 minutes.
91:03 Humming for 10 minutes. 10 minutes. Both sides.
91:05 10 minutes. Both sides. What do you mean by humming? Give me an
91:06 What do you mean by humming? Give me an example.
91:06 example. Oh, humming. Oh, humming. Oh, yeah. That
91:09 Oh, humming. Oh, humming. Oh, yeah. That sound is transmitted
91:12 sound is transmitted to the vagus nerve. So, it's like
91:14 to the vagus nerve. So, it's like shaking that vagus nerve and it creates
91:16 shaking that vagus nerve and it creates electrical impulses uh in the vagus
91:18 electrical impulses uh in the vagus nerve. That's been shown. Singing,
91:20 nerve. That's been shown. Singing, singing, singing. humming,
91:23 singing, singing. humming, laughing.
91:24 laughing. You know that there are laughing clubs.
91:27 You know that there are laughing clubs. Maybe you should start a laughing club.
91:29 Maybe you should start a laughing club. Laughing clubs. Yeah, laugh for no
91:31 Laughing clubs. Yeah, laugh for no reason. Just sit there and laugh. So
91:33 reason. Just sit there and laugh. So that diaphragmatic movement, that
91:35 that diaphragmatic movement, that constant diaphragmatic movement in
91:37 constant diaphragmatic movement in laughing stimulates the vagus nerve.
91:39 laughing stimulates the vagus nerve. I heard you talk about this valva
91:41 I heard you talk about this valva maneuver.
91:42 maneuver. Yeah.
91:43 Yeah. What's that?
91:43 What's that? The valva is when you breathe in
91:46 The valva is when you breathe in and then you breathe out, but don't let
91:48 and then you breathe out, but don't let the air out and you're straining. And
91:51 the air out and you're straining. And that straining compresses the thoracic
91:53 that straining compresses the thoracic cavity but also the abdominal cavity and
91:55 cavity but also the abdominal cavity and stimulates the vagus nerve. And that is
91:58 stimulates the vagus nerve. And that is also found to be very very helpful.
92:00 also found to be very very helpful. And remind me again if I have a healthy
92:02 And remind me again if I have a healthy vagus nerve, if I have a calm healthy
92:05 vagus nerve, if I have a calm healthy vagus nerve, what is the downstream
92:07 vagus nerve, what is the downstream impact going to be across my life?
92:09 impact going to be across my life? Downstream impact is going to be faster
92:11 Downstream impact is going to be faster healing,
92:12 healing, less coronary disease, less high blood
92:14 less coronary disease, less high blood pressure. Your blood will not be so
92:16 pressure. Your blood will not be so sticky and make blood clots less
92:20 sticky and make blood clots less inflammatory markers.
92:23 inflammatory markers. The blood tests that we do, the
92:24 The blood tests that we do, the interucan 6, the tumor necrosis factor,
92:26 interucan 6, the tumor necrosis factor, the CRP levels, small dense LDL, we
92:30 the CRP levels, small dense LDL, we didn't even talk about LDL, but LDL, the
92:32 didn't even talk about LDL, but LDL, the bad cholesterol, all those get better.
92:35 bad cholesterol, all those get better. And what is the the the role of LDL as
92:38 And what is the the the role of LDL as it relates to heart disease? Because
92:40 it relates to heart disease? Because I've heard this word cholesterol. I
92:41 I've heard this word cholesterol. I really don't really understand what it
92:43 really don't really understand what it is. or I know there's good cholesterol,
92:44 is. or I know there's good cholesterol, there's bad cholesterol. I I guess that
92:46 there's bad cholesterol. I I guess that bad cholesterol comes from fast food and
92:49 bad cholesterol comes from fast food and processed foods and stuff, but simply
92:51 processed foods and stuff, but simply what do I need to understand about the
92:53 what do I need to understand about the role cholesterol plays in heart disease
92:54 role cholesterol plays in heart disease and how to not have bad cholesterol?
92:58 and how to not have bad cholesterol? The LDL car carries most of the
93:00 The LDL car carries most of the cholesterol in the body. It's not
93:02 cholesterol in the body. It's not complicated.
93:03 complicated. LDL is what
93:04 LDL is what is LDL is a lipoprotein.
93:07 is LDL is a lipoprotein. Yeah.
93:07 Yeah. Right. And you have HDL which is good
93:10 Right. And you have HDL which is good cholesterol. LDL, bad cholesterol, total
93:12 cholesterol. LDL, bad cholesterol, total cholesterol and triglycerides. Okay,
93:14 cholesterol and triglycerides. Okay, these are the four tests.
93:15 these are the four tests. So the LDL is a reflection of your
93:17 So the LDL is a reflection of your cholesterol. Now cholesterol is a normal
93:21 cholesterol. Now cholesterol is a normal molecule in your body and you should
93:25 molecule in your body and you should have cholesterol because if you don't
93:26 have cholesterol because if you don't have cholesterol, I don't think you live
93:28 have cholesterol, I don't think you live too long and you need to move this
93:30 too long and you need to move this cholesterol around your body. Every cell
93:32 cholesterol around your body. Every cell in the body makes cholesterol. Your
93:34 in the body makes cholesterol. Your liver makes most of the cholesterol
93:35 liver makes most of the cholesterol actually. Now what happens is that when
93:38 actually. Now what happens is that when that LDL molecule becomes damaged, it
93:41 that LDL molecule becomes damaged, it becomes a small dense LDL particle. A
93:45 becomes a small dense LDL particle. A damaged LDL causes inflammation because
93:48 damaged LDL causes inflammation because now you have a molecule that's floating
93:50 now you have a molecule that's floating around your body. It's been damaged.
93:52 around your body. It's been damaged. It's small. It's dense. Now your white
93:56 It's small. It's dense. Now your white cells, your macrofasages see that and
93:59 cells, your macrofasages see that and want to gobble it up. That's number one.
94:01 want to gobble it up. That's number one. Number two, these molecules get oxidized
94:03 Number two, these molecules get oxidized in the lungs and then they the first
94:06 in the lungs and then they the first place they come out from after the lungs
94:08 place they come out from after the lungs is the coronary arteries. So it's the
94:10 is the coronary arteries. So it's the small dense LDL particles which are now
94:14 small dense LDL particles which are now oxidized
94:15 oxidized they destroy the glycoalix which is the
94:18 they destroy the glycoalix which is the lining of the coronary arteries and they
94:20 lining of the coronary arteries and they activate the endothelium.
94:22 activate the endothelium. When they activate the endothelium which
94:24 When they activate the endothelium which is the lining of your coronary arteries
94:27 is the lining of your coronary arteries that's when you're going to start
94:28 that's when you're going to start getting aosclerosis. So atherosclerosis
94:32 getting aosclerosis. So atherosclerosis and LDL are related but it's not the the
94:35 and LDL are related but it's not the the total cholesterol. It's when you have
94:37 total cholesterol. It's when you have small dense damaged LDL. So let me
94:41 small dense damaged LDL. So let me explain the impact in my practice.
94:44 explain the impact in my practice. Patients come to me because they either
94:47 Patients come to me because they either cannot tolerate cholesterol medications
94:49 cannot tolerate cholesterol medications or they don't want to take cholesterol
94:50 or they don't want to take cholesterol medications. What I do then is I look
94:53 medications. What I do then is I look and say okay your LDL
94:56 and say okay your LDL is it high or low? Is it is that what
94:58 is it high or low? Is it is that what I'm really concerned about or is it that
95:00 I'm really concerned about or is it that it's small dense LDL? So I do a
95:02 it's small dense LDL? So I do a subfractionation and this is important
95:04 subfractionation and this is important for you to understand because they don't
95:07 for you to understand because they don't do it in every country in the world.
95:08 do it in every country in the world. It's very difficult to get some of these
95:09 It's very difficult to get some of these blood tests abroad. But in the United
95:11 blood tests abroad. But in the United States is easy to do it. It's called
95:13 States is easy to do it. It's called subfraction of your LDL. So it tells you
95:16 subfraction of your LDL. So it tells you whether you have small dense LDL or not.
95:18 whether you have small dense LDL or not. So let's say you came to me and your LDL
95:20 So let's say you came to me and your LDL is 150. But if it's all made up of
95:24 is 150. But if it's all made up of large, fluffy, good, normal LDL, it's
95:30 large, fluffy, good, normal LDL, it's not going to be implicated in your
95:31 not going to be implicated in your inflammation or in your coronary artery
95:33 inflammation or in your coronary artery disease. I leave that alone. But if you
95:37 disease. I leave that alone. But if you have small dense LDL, that also is a
95:40 have small dense LDL, that also is a problem because it's pointing me towards
95:43 problem because it's pointing me towards inflammation. Because what causes small,
95:45 inflammation. Because what causes small, dense, damaged LDL are glucose, omega6,
95:50 dense, damaged LDL are glucose, omega6, advanced glycation end products, toxins,
95:54 advanced glycation end products, toxins, and lipopolyaccharides that come from
95:56 and lipopolyaccharides that come from the gut, leaky gut. So now I just gave
96:00 the gut, leaky gut. So now I just gave you the primer
96:02 you the primer for treating coronary artery disease.
96:05 for treating coronary artery disease. These five things
96:08 These five things promote coronary artery disease and it's
96:10 promote coronary artery disease and it's small dense LDL. Those macrofasages they
96:14 small dense LDL. Those macrofasages they engulf small dense LDL and those
96:17 engulf small dense LDL and those macrofasages become foam cells. The
96:19 macrofasages become foam cells. The endothelium has been already activated.
96:23 endothelium has been already activated. So they
96:25 So they bind to the endothelial lining.
96:27 bind to the endothelial lining. What's the endothelium?
96:28 What's the endothelium? The endothelium is the lining of the
96:29 The endothelium is the lining of the artery in your coronary artery. So these
96:32 artery in your coronary artery. So these white cells that are full of fat, the
96:34 white cells that are full of fat, the bad fat attach themselves to it and then
96:37 bad fat attach themselves to it and then become endothelialized. That means they
96:39 become endothelialized. That means they become part of the wall.
96:40 become part of the wall. So they become a plaque.
96:41 So they become a plaque. That's now you got it. That's making the
96:44 That's now you got it. That's making the plaque. You just hit it right on the
96:45 plaque. You just hit it right on the head.
96:46 head. So how would I naturally lower my LDL
96:49 So how would I naturally lower my LDL without taking statins or any of these
96:51 without taking statins or any of these other medications? Is there a way to to
96:53 other medications? Is there a way to to do it naturally?
96:55 do it naturally? You you stay away from from meat. Yes.
96:57 You you stay away from from meat. Yes. You've gone on a vegetarian diet. Yes,
96:59 You've gone on a vegetarian diet. Yes, it'll come down. But there's no need to
97:01 it'll come down. But there's no need to or meat.
97:02 or meat. Or meat because they'll all have
97:03 Or meat because they'll all have cholesterol.
97:04 cholesterol. Even chicken.
97:04 Even chicken. Even chicken. Yeah. If you want to bring
97:06 Even chicken. Yeah. If you want to bring it down. But the question is, do you
97:08 it down. But the question is, do you really want to bring it down? Do you
97:09 really want to bring it down? Do you really want to bring it down? Later. The
97:11 really want to bring it down? Later. The most recent data is very scary. It shows
97:14 most recent data is very scary. It shows that patients who have the highest LDLs
97:16 that patients who have the highest LDLs actually make it into the '9s and live a
97:19 actually make it into the '9s and live a healthier life. It's not the LDL, it's
97:22 healthier life. It's not the LDL, it's the damaged LDL that's the problem. So
97:25 the damaged LDL that's the problem. So this the medical profession as a whole
97:28 this the medical profession as a whole and all of us are having a problem
97:30 and all of us are having a problem grappling with this. Is it the total LDL
97:32 grappling with this. Is it the total LDL that we should be concerned about? Point
97:35 that we should be concerned about? Point to your question. What is a normal
97:37 to your question. What is a normal cholesterol and should we be lowering
97:39 cholesterol and should we be lowering cholesterol and the answer really is not
97:41 cholesterol and the answer really is not that simple. What we should be doing is
97:44 that simple. What we should be doing is not to have small dense LDL. So the
97:47 not to have small dense LDL. So the question is that's the issue
97:49 question is that's the issue and small dense LDL five things. Sugar
97:52 and small dense LDL five things. Sugar causes it. Omega6,
97:54 causes it. Omega6, advanced glycation end products, toxins,
97:56 advanced glycation end products, toxins, and leaky gut. They make your
97:58 and leaky gut. They make your cholesterol become small, dense
98:00 cholesterol become small, dense particles.
98:01 particles. A lot of people are concerned about
98:02 A lot of people are concerned about eating eggs because they think it might
98:03 eating eggs because they think it might raise their cholesterol levels.
98:05 raise their cholesterol levels. Absolutely no concerns whatsoever.
98:07 Absolutely no concerns whatsoever. And what about statins? So a lot of
98:09 And what about statins? So a lot of people are prescribed statins when they
98:10 people are prescribed statins when they have heart related issues. Are they safe
98:13 have heart related issues. Are they safe in the long run? Are they effective?
98:16 in the long run? Are they effective? Okay, first part, are they safe? For the
98:19 Okay, first part, are they safe? For the most part, they are. But at least 20 to
98:21 most part, they are. But at least 20 to 30% of patients will suffer from
98:23 30% of patients will suffer from psychopenia which is loss of muscles. So
98:25 psychopenia which is loss of muscles. So they'll get weakness, aches, pains.
98:29 they'll get weakness, aches, pains. They'll get diabetes because it causes
98:32 They'll get diabetes because it causes mitochondrial dysfunction. So they'll de
98:35 mitochondrial dysfunction. So they'll de sugar problems and many of them also do
98:38 sugar problems and many of them also do developmental diseases. So I've seen
98:40 developmental diseases. So I've seen that they become forgetful and they just
98:42 that they become forgetful and they just can't think right and I stop the statins
98:44 can't think right and I stop the statins and all of a sudden they start feeling
98:46 and all of a sudden they start feeling better. So no, not all statins are safe.
98:50 better. So no, not all statins are safe. You need to monitor statins also. They
98:52 You need to monitor statins also. They can cause liver dysfunction. So you need
98:54 can cause liver dysfunction. So you need to watch that. Number two is that the
98:57 to watch that. Number two is that the statins are they effective. Effective in
99:00 statins are they effective. Effective in what? If the end point is to reduce your
99:04 what? If the end point is to reduce your LDL. Yeah, statins will bring your LDL
99:07 LDL. Yeah, statins will bring your LDL down. But does that translate to a
99:10 down. But does that translate to a decrease in cardiovascular event rates?
99:12 decrease in cardiovascular event rates? That's a totally different question. You
99:14 That's a totally different question. You see? So, is your goal just to reduce the
99:17 see? So, is your goal just to reduce the LDL or is your goal to reduce the
99:20 LDL or is your goal to reduce the damaged, oxidized, small, dense LDL
99:23 damaged, oxidized, small, dense LDL levels? And when people say they have
99:25 levels? And when people say they have high blood pressure, that's because
99:28 high blood pressure, that's because there's potentially a narrowing of their
99:31 there's potentially a narrowing of their blood vessels. So, the the the blood has
99:34 blood vessels. So, the the the blood has less space to go through. So, it's kind
99:36 less space to go through. So, it's kind of like squeezing a host pipe that it
99:37 of like squeezing a host pipe that it gets it gets shoots out faster. Is that
99:39 gets it gets shoots out faster. Is that typically what it means by high blood
99:40 typically what it means by high blood pressure?
99:41 pressure? Sort of. Let me explain. When your
99:44 Sort of. Let me explain. When your insulin levels are running high, all
99:46 insulin levels are running high, all your blood vessels and your capillaries
99:48 your blood vessels and your capillaries all don't vasoddilate properly.
99:51 all don't vasoddilate properly. Oh, okay. And vasoddilation is the
99:53 Oh, okay. And vasoddilation is the expansion and shrinking.
99:54 expansion and shrinking. Yeah. So, they don't vasod dial
99:56 Yeah. So, they don't vasod dial properly. So, you will get high blood
99:57 properly. So, you will get high blood pressure. So, one of the biggest things
99:59 pressure. So, one of the biggest things I've noticed with diabetes patients is
100:01 I've noticed with diabetes patients is when I bring the insulin levels down
100:02 when I bring the insulin levels down through my fasting program and dietary
100:04 through my fasting program and dietary changes, the blood pressures go away.
100:06 changes, the blood pressures go away. See, that's another thing that there's
100:08 See, that's another thing that there's no such thing as essential hypertension.
100:11 no such thing as essential hypertension. Essential hypertention meaning that you
100:14 Essential hypertention meaning that you just have high blood pressure. There's
100:16 just have high blood pressure. There's always a cause. Either you have sleep
100:17 always a cause. Either you have sleep apnea or you have hyperinsulinemia
100:19 apnea or you have hyperinsulinemia because sleep apnea also causes
100:21 because sleep apnea also causes hypertension. So hypertension does not
100:24 hypertension. So hypertension does not always necessarily mean that you're
100:25 always necessarily mean that you're going to be committed to blood pressure
100:26 going to be committed to blood pressure medications for the rest of your life.
100:28 medications for the rest of your life. I've heard you talk quite a lot about
100:30 I've heard you talk quite a lot about breakfast. People ask you about what
100:32 breakfast. People ask you about what they should have for breakfast because
100:33 they should have for breakfast because everybody wants to know um how they
100:35 everybody wants to know um how they should be starting their day. What is
100:37 should be starting their day. What is your advice on what we should be eating
100:39 your advice on what we should be eating for breakfast to have a healthy heart
100:40 for breakfast to have a healthy heart and cardiovascular system?
100:44 and cardiovascular system? Breakfast is a tough one. I tell most of
100:46 Breakfast is a tough one. I tell most of my patients that if you absolutely have
100:48 my patients that if you absolutely have to have a breakfast, have a couple of
100:49 to have a breakfast, have a couple of eggs and if you are a vegetarian, it
100:54 eggs and if you are a vegetarian, it makes it a little bit more tough. Um,
100:56 makes it a little bit more tough. Um, but you can have some kefir with two to
100:59 but you can have some kefir with two to three blueberries, two to three
101:00 three blueberries, two to three blackberries, and about four blueberries
101:03 blackberries, and about four blueberries in it. And then you put your inulin
101:06 in it. And then you put your inulin powder in it and that's your breakfast.
101:08 powder in it and that's your breakfast. That's all you should have. But a heavy
101:10 That's all you should have. But a heavy breakfast and also breakfast in my
101:13 breakfast and also breakfast in my opinion breakfast should be we should
101:16 opinion breakfast should be we should pass breakfast for most adults and just
101:19 pass breakfast for most adults and just go for lunch and dinner only cuz then
101:21 go for lunch and dinner only cuz then you can do your 18 six. If you
101:24 you can do your 18 six. If you absolutely going to have breakfast then
101:25 absolutely going to have breakfast then you have breakfast and lunch and then no
101:27 you have breakfast and lunch and then no dinner. But you got to plan 18 hours. 18
101:31 dinner. But you got to plan 18 hours. 18 hours is optimal and we can do it. A lot
101:33 hours is optimal and we can do it. A lot of people say they can do it, but most
101:35 of people say they can do it, but most of us can do it. You will notice how
101:37 of us can do it. You will notice how much more energy you have. You know,
101:39 much more energy you have. You know, when I do my OMAD, which I do at least 3
101:42 when I do my OMAD, which I do at least 3 to four times a week, which I only have
101:44 to four times a week, which I only have one meal a day, at 6:00 in the evening,
101:47 one meal a day, at 6:00 in the evening, I actually have more energy than I do in
101:49 I actually have more energy than I do in the morning.
101:50 the morning. What does your diet look like, doctor?
101:52 What does your diet look like, doctor? My diet? Yeah,
101:54 My diet? Yeah, I do eat meat and I do eat fish,
101:57 I do eat meat and I do eat fish, chicken, and turkey and but I'll have I
102:00 chicken, and turkey and but I'll have I source it right. Always make sure that
102:02 source it right. Always make sure that it's grass-finished. Always make sure
102:04 it's grass-finished. Always make sure it's organic and I'll have small amounts
102:05 it's organic and I'll have small amounts of it.
102:06 of it. Grass-fed, grass-fed.
102:08 Grass-fed, grass-fed. Grass finished.
102:09 Grass finished. What does that mean?
102:11 What does that mean? Grass-fed just means they took it out
102:12 Grass-fed just means they took it out one day and put it out in the field and
102:15 one day and put it out in the field and they fed it some grass. So, this
102:17 they fed it some grass. So, this advertising that industry does is a
102:20 advertising that industry does is a little deceiving.
102:21 little deceiving. And I had to investigate this to find
102:23 And I had to investigate this to find out. It's a lie. It has to be grass-fed
102:26 out. It's a lie. It has to be grass-fed from beginning to end. So then it's
102:29 from beginning to end. So then it's going to have more omega-3 in it. It'll
102:30 going to have more omega-3 in it. It'll have more K2 in it. It'll have more
102:32 have more K2 in it. It'll have more nutrition and have less toxins also in
102:34 nutrition and have less toxins also in it. So less omega6 because if your cows
102:38 it. So less omega6 because if your cows are eating corn,
102:41 are eating corn, then you're going to get all that
102:42 then you're going to get all that omega6. And the cow is not supposed to
102:44 omega6. And the cow is not supposed to be eating corn. Cow is supposed to be
102:46 be eating corn. Cow is supposed to be eating grass.
102:48 eating grass. So I do eat some meats and I eat some
102:50 So I do eat some meats and I eat some vegetables with it. Coming from a
102:53 vegetables with it. Coming from a background, my great-grandfather's from
102:55 background, my great-grandfather's from India. We do eat Indian food as well. So
102:57 India. We do eat Indian food as well. So we have some lentils
103:00 we have some lentils and some curries of course. So we can
103:02 and some curries of course. So we can cook the meats in a in a curry form as
103:04 cook the meats in a in a curry form as well. I don't eat much chapatis because
103:07 well. I don't eat much chapatis because those are very full of carbs. I love the
103:11 those are very full of carbs. I love the taste of it. I love naan once in a
103:13 taste of it. I love naan once in a while.
103:13 while. And what do you supplement with? What's
103:15 And what do you supplement with? What's your supplement stack look like? I take
103:16 your supplement stack look like? I take D3, K2,
103:19 D3, K2, omega3 fish oil. So I take two two
103:22 omega3 fish oil. So I take two two capsules every day. Vitamin C 1 g inulin
103:27 capsules every day. Vitamin C 1 g inulin kefir. I drink that every day and I
103:29 kefir. I drink that every day and I count it. Magnesium
103:32 count it. Magnesium and I take nattokynise.
103:36 and I take nattokynise. Nattokynise. Nattokynise I take about
103:39 Nattokynise. Nattokynise I take about 8,000 units a day. What it is is that
103:44 8,000 units a day. What it is is that it does thin the blood a little bit so
103:46 it does thin the blood a little bit so that you're less likely to make blood
103:48 that you're less likely to make blood clots. Part of metabolic syndrome is a
103:50 clots. Part of metabolic syndrome is a tendency to make blood clots and have a
103:52 tendency to make blood clots and have a high fibbrin level. My frigen level was
103:54 high fibbrin level. My frigen level was at the upper limits of normal. So I just
103:56 at the upper limits of normal. So I just take nattokinise. It doesn't cause any
103:58 take nattokinise. It doesn't cause any bleeding or any problems like that. And
104:01 bleeding or any problems like that. And then I also take a probiotic and I it's
104:05 then I also take a probiotic and I it's it's basically is called mega spores
104:10 it's basically is called mega spores probiotic. So there's it's spores.
104:15 probiotic. So there's it's spores. There's no real bacteria in there. It's
104:16 There's no real bacteria in there. It's all spores.
104:17 all spores. What's a spore?
104:18 What's a spore? A spore a bacteria
104:21 A spore a bacteria can survive
104:23 can survive by sporulating. So it becomes a spore.
104:25 by sporulating. So it becomes a spore. So it's no longer a bacteria, it's a
104:27 So it's no longer a bacteria, it's a spore, but it'll germinate under the
104:29 spore, but it'll germinate under the right circumstances. So these little
104:32 right circumstances. So these little things, they have they are the spores of
104:35 things, they have they are the spores of the bacteria that are in soil. So soil
104:39 the bacteria that are in soil. So soil has certain species of bacteria, the
104:41 has certain species of bacteria, the basilus species.
104:43 basilus species. The spores
104:46 The spores I consume, they go in my gut because
104:48 I consume, they go in my gut because remember if I take a probiotic,
104:51 remember if I take a probiotic, the acidity in my stomach is going to
104:53 the acidity in my stomach is going to kill most of the bacteria.
104:56 kill most of the bacteria. So what does a spore do? Sorry.
104:57 So what does a spore do? Sorry. So the spore passes through the stomach,
105:00 So the spore passes through the stomach, goes all the way down to your intestines
105:02 goes all the way down to your intestines and then it germinates there. It grows
105:05 and then it germinates there. It grows there. So now the spore becomes a
105:06 there. So now the spore becomes a bacteria.
105:07 bacteria. Okay?
105:08 Okay? So it gets past that acidity of your
105:11 So it gets past that acidity of your stomach. So, I do take that as a
105:13 stomach. So, I do take that as a supplement as well.
105:14 supplement as well. Dr. Jay, what is the most important
105:15 Dr. Jay, what is the most important thing we didn't talk about that we
105:17 thing we didn't talk about that we should have talked about for the people
105:19 should have talked about for the people at home that have clicked on this
105:21 at home that have clicked on this episode? And you know, I guess one way
105:23 episode? And you know, I guess one way that you might tackle this question is
105:24 that you might tackle this question is just by thinking about the most popular
105:27 just by thinking about the most popular questions you get asked from your
105:28 questions you get asked from your audience on a daily basis that we might
105:31 audience on a daily basis that we might not have covered today.
105:33 not have covered today. How can I screen myself right now? What
105:36 How can I screen myself right now? What test should I do right now to see
105:41 test should I do right now to see at what stage of inflammation and
105:43 at what stage of inflammation and coronary artery disease am I in? Because
105:45 coronary artery disease am I in? Because coronary arteries is the number one
105:46 coronary arteries is the number one cause of death and inflammation is the
105:48 cause of death and inflammation is the underlying cause. So what test should I
105:51 underlying cause. So what test should I be doing right now? That is an important
105:53 be doing right now? That is an important question. If you're over the age of 30
105:56 question. If you're over the age of 30 and you have concerns, one, get a
105:59 and you have concerns, one, get a coronary calcium score.
106:02 coronary calcium score. If your coronary calcium score is zero,
106:05 If your coronary calcium score is zero, you have no calcium,
106:07 you have no calcium, then you're in a good place. If you have
106:10 then you're in a good place. If you have coronary calcium,
106:12 coronary calcium, you need to go see a good cardiologist
106:14 you need to go see a good cardiologist that's going to do a prevention program
106:15 that's going to do a prevention program because it means you already have
106:16 because it means you already have athoscerosis.
106:18 athoscerosis. Athoscerosis meaning plaque
106:19 Athoscerosis meaning plaque hardening of the arteries.
106:21 hardening of the arteries. Yeah.
106:21 Yeah. Number two, you need a good blood test
106:25 Number two, you need a good blood test to look for inflammation. The blood test
106:27 to look for inflammation. The blood test that I order is called a Cleveland heart
106:30 that I order is called a Cleveland heart labs. In it you get your HDL, LDL,
106:35 labs. In it you get your HDL, LDL, cholesterol. But you look at particle
106:37 cholesterol. But you look at particle sizes.
106:39 sizes. Is my LDL small dense? If it is small
106:42 Is my LDL small dense? If it is small dense particles, is it oxidized? Yes, I
106:45 dense particles, is it oxidized? Yes, I have a problem. My CRP level,
106:49 have a problem. My CRP level, other inflammatory interucan 6,
106:51 other inflammatory interucan 6, timonucosis factor, hemoglobin A1C,
106:56 timonucosis factor, hemoglobin A1C, sugar level,
106:58 sugar level, all these are part of that panel of
107:00 all these are part of that panel of tests. So you should get a good
107:03 tests. So you should get a good inflammatory panel which is the
107:05 inflammatory panel which is the Cleveland Heart Labs. So those are the
107:07 Cleveland Heart Labs. So those are the two things that everyone must do. Get a
107:09 two things that everyone must do. Get a coronary calcium score all adults and do
107:12 coronary calcium score all adults and do an inflammatory panel. Inflammation you
107:15 an inflammatory panel. Inflammation you got to know whether you look you know
107:17 got to know whether you look you know you may not feel inflammation. It's not
107:20 you may not feel inflammation. It's not like you get a big pimple and it's going
107:21 like you get a big pimple and it's going to hurt you but if you have inflammation
107:23 to hurt you but if you have inflammation in the body yeah you could know about
107:25 in the body yeah you could know about it. How you have mental fog? I'm not
107:28 it. How you have mental fog? I'm not optimal doc. My memor is going down. I'm
107:32 optimal doc. My memor is going down. I'm depressed. Depression is inflammation.
107:35 depressed. Depression is inflammation. Depression is not something
107:36 Depression is not something psychological. Okay? Now, this I have to
107:39 psychological. Okay? Now, this I have to stress to you and I tell all my
107:40 stress to you and I tell all my patients, you come in, I'm depressed.
107:42 patients, you come in, I'm depressed. You're depressed because
107:45 You're depressed because that very symptom of depression is a
107:47 that very symptom of depression is a symptom of your inflammation.
107:50 symptom of your inflammation. You can't be depressed about something
107:52 You can't be depressed about something that oh my car is not working very well.
107:55 that oh my car is not working very well. That's a difference from depression.
107:58 That's a difference from depression. Depression is is is inflammation in the
108:00 Depression is is is inflammation in the brain. So you'll be surprised how many
108:03 brain. So you'll be surprised how many patients go on my anti-inflammatory
108:04 patients go on my anti-inflammatory diet, which is I change the diet and fix
108:06 diet, which is I change the diet and fix them all up and the depression goes
108:08 them all up and the depression goes away. So ask yourself not just questions
108:12 away. So ask yourself not just questions about physically how you're feeling but
108:14 about physically how you're feeling but mentally also.
108:17 mentally also. And if the answer is that doc I'm not
108:19 And if the answer is that doc I'm not optimal I'm forgetful I'm depressed I'm
108:22 optimal I'm forgetful I'm depressed I'm having sleep dis disturbance I'm
108:24 having sleep dis disturbance I'm relation things my I've changed my body
108:27 relation things my I've changed my body is also you have inflammation
108:30 is also you have inflammation we have a closing tradition on this
108:33 we have a closing tradition on this podcast where the last guest leaves a
108:34 podcast where the last guest leaves a question for the next guest not knowing
108:35 question for the next guest not knowing who they're leaving it for and the
108:37 who they're leaving it for and the question that's been left for you is
108:38 question that's been left for you is what was the most difficult day of your
108:41 what was the most difficult day of your life and how did you overcome it
108:43 life and how did you overcome it the most difficult day of my life
108:46 the most difficult day of my life was was a very personal one for me, you
108:49 was was a very personal one for me, you know, when when when when my my dad
108:51 know, when when when when my my dad passed away and you know, and I'm Dr.
108:55 passed away and you know, and I'm Dr. Jay
108:57 Jay and uh
108:59 and uh and when he he was sick and he didn't
109:02 and when he he was sick and he didn't want to go to the hospital
109:05 want to go to the hospital and then he passed away in his sleep and
109:08 and then he passed away in his sleep and then you always ask yourself, could I
109:10 then you always ask yourself, could I have done something differently? you
109:12 have done something differently? you know you second guess yourself and that
109:15 know you second guess yourself and that second guessing happened with him of
109:16 second guessing happened with him of course it was very very painful but it
109:19 course it was very very painful but it happens with patients as well but how
109:23 happens with patients as well but how did I overcome it it taught me a lot
109:26 did I overcome it it taught me a lot about my own limitations as well as a
109:28 about my own limitations as well as a physician as a son as a scientist as a
109:32 physician as a son as a scientist as a reader
109:33 reader and that's why I'm motivated to do what
109:36 and that's why I'm motivated to do what we are doing here also is to do the best
109:38 we are doing here also is to do the best you can to educate people What was he
109:41 you can to educate people What was he sick with? What was his predicament?
109:44 sick with? What was his predicament? He was a cardiac patient already and
109:46 He was a cardiac patient already and he's he's got a very interesting story
109:48 he's he's got a very interesting story actually. He was told that he only had a
109:50 actually. He was told that he only had a few years to live after he had bypass
109:52 few years to live after he had bypass surgery. But then I put him through all
109:55 surgery. But then I put him through all the things we talked about today. He
109:57 the things we talked about today. He lost a lot of weight. He went from 185
109:59 lost a lot of weight. He went from 185 lbs to 135 lbs. He changed his diet and
110:01 lbs to 135 lbs. He changed his diet and he lived another 30 years and he did
110:04 he lived another 30 years and he did another six world tours and traveled and
110:06 another six world tours and traveled and he had a wonderful life. Okay. the
110:09 he had a wonderful life. Okay. the impact of simple things that we can do
110:11 impact of simple things that we can do to change his life. So he stopped
110:13 to change his life. So he stopped drinking all alcohol as well and he he
110:15 drinking all alcohol as well and he he really had a very clean diet and
110:17 really had a very clean diet and everything else. But that night he came
110:20 everything else. But that night he came down with some sort of infection. He had
110:22 down with some sort of infection. He had massive diarrhea and massive massive
110:25 massive diarrhea and massive massive explosion. And I think that what really
110:27 explosion. And I think that what really happened is he he got overwhelmed. You
110:29 happened is he he got overwhelmed. You see it's the gut. He got overwhelmed
110:31 see it's the gut. He got overwhelmed with a toxin from his gut and then he
110:35 with a toxin from his gut and then he passed away. But but here's a man who
110:39 passed away. But but here's a man who who lived to 89 who was supposed to have
110:42 who lived to 89 who was supposed to have passed away in his 60s and the impact
110:45 passed away in his 60s and the impact but but the thing is that you know we we
110:47 but but the thing is that you know we we we shouldn't second guessess ourselves.
110:49 we shouldn't second guessess ourselves. We don't want to live in the past.
110:52 We don't want to live in the past. I want to live in the present moment not
110:54 I want to live in the present moment not even in the future.
110:57 even in the future. So all these things have taught me to
110:59 So all these things have taught me to live in the moment. And one of the
111:02 live in the moment. And one of the skills I've developed is just that that
111:05 skills I've developed is just that that when I'm with you, I'm only with you. If
111:08 when I'm with you, I'm only with you. If I'm doing your surgery, I'm only doing
111:10 I'm doing your surgery, I'm only doing your surgery. If I'm having a
111:11 your surgery. If I'm having a conversation with you, you and I are the
111:14 conversation with you, you and I are the only people that matter right now. And
111:16 only people that matter right now. And this being in the moment, that that
111:19 this being in the moment, that that ability to be that is something I
111:22 ability to be that is something I learned over time because before you
111:24 learned over time because before you know something bad happens, your next
111:26 know something bad happens, your next case, you're still reminiscing about
111:28 case, you're still reminiscing about that and you're regretting that. You
111:30 that and you're regretting that. You can't do that. Life is only expressed in
111:33 can't do that. Life is only expressed in this moment right now. So learning how
111:36 this moment right now. So learning how to live in the moment applies not only
111:38 to live in the moment applies not only to me but to everybody. We all living in
111:41 to me but to everybody. We all living in the past otherwise and or we're worried
111:45 the past otherwise and or we're worried about the future. So how do you learn
111:48 about the future. So how do you learn present moment awareness and live in
111:50 present moment awareness and live in this moment so that we will be happier.
111:54 this moment so that we will be happier. We can do what we want to do. Make the
111:56 We can do what we want to do. Make the changes we need to make. get the courage
111:58 changes we need to make. get the courage and make the and have the willpower to
112:01 and make the and have the willpower to go out and make a difference to yourself
112:03 go out and make a difference to yourself and to everyone around you starts with
112:05 and to everyone around you starts with you being here. Are you here or are you
112:09 you being here. Are you here or are you in your past or have you gone off into
112:12 in your past or have you gone off into the future? That's a skill that medicine
112:16 the future? That's a skill that medicine has taught me and I've become humbled by
112:18 has taught me and I've become humbled by it. And this is something that I have
112:22 it. And this is something that I have learned and I want to pass on to all my
112:25 learned and I want to pass on to all my patients too as well that okay you've
112:26 patients too as well that okay you've got all these things going on but learn
112:29 got all these things going on but learn to live in the moment.
112:31 to live in the moment. Dr. Jay, thank you so much. Thank you so
112:33 Dr. Jay, thank you so much. Thank you so much for the wisdom that you've shared
112:34 much for the wisdom that you've shared with so many people. If people aren't
112:35 with so many people. If people aren't familiar with you, you're extremely
112:37 familiar with you, you're extremely prolific and well loved across the
112:39 prolific and well loved across the internet for the the public education
112:42 internet for the the public education work you've done and the the heightening
112:44 work you've done and the the heightening of awareness of people's heart and their
112:46 of awareness of people's heart and their cardiovascular health more broadly. And
112:47 cardiovascular health more broadly. And until really before you started making
112:49 until really before you started making videos online, a lot of this stuff was
112:52 videos online, a lot of this stuff was opaque. It was a mystery to a lot of
112:54 opaque. It was a mystery to a lot of people. And your YouTube channel has
112:56 people. And your YouTube channel has almost a million subscribers now. And
112:58 almost a million subscribers now. And you you go through some of these
112:59 you you go through some of these subjects that we've talked about today
113:01 subjects that we've talked about today in even greater detail. So I'd highly
113:03 in even greater detail. So I'd highly recommend people go and check you out
113:04 recommend people go and check you out there. But also just thank you for
113:05 there. But also just thank you for saving so many people's lives cuz you
113:07 saving so many people's lives cuz you know that's what you do day in day out
113:08 know that's what you do day in day out and that's an incredibly I mean there's
113:10 and that's an incredibly I mean there's probably not a higher calling one can
113:12 probably not a higher calling one can answer. So thank you for doing what you
113:14 answer. So thank you for doing what you do and I can see now why people love
113:16 do and I can see now why people love you. so much on online and on YouTube
113:18 you. so much on online and on YouTube because you're so incredibly engaging, I
113:21 because you're so incredibly engaging, I guess is the word, engaging, wise, and
113:24 guess is the word, engaging, wise, and you make the information accessible in a
113:26 you make the information accessible in a way that I don't think has been done
113:28 way that I don't think has been done before. So, thank you so much. It's a
113:29 before. So, thank you so much. It's a pleasure to meet you and to have this
113:30 pleasure to meet you and to have this conversation with you, and thank you for
113:31 conversation with you, and thank you for enlightening my audience.
113:32 enlightening my audience. The pleasure was mine. Thank you.
113:35 The pleasure was mine. Thank you. This has always blown my mind a little
113:36 This has always blown my mind a little bit. 53% of you that listen to the show
113:39 bit. 53% of you that listen to the show regularly haven't yet subscribed to the
113:41 regularly haven't yet subscribed to the show. So, could I ask you for a favor?
113:43 show. So, could I ask you for a favor? If you like the show and you like what
113:44 If you like the show and you like what we do here and you want to support us,
113:45 we do here and you want to support us, the free simple way that you can do just
113:47 the free simple way that you can do just that is by hitting the subscribe button.
113:49 that is by hitting the subscribe button. And my commitment to you is if you do
113:51 And my commitment to you is if you do that, then I'll do everything in my
113:52 that, then I'll do everything in my power, me and my team, to make sure that
113:54 power, me and my team, to make sure that this show is better for you every single
113:56 this show is better for you every single week. We'll listen to your feedback.
113:58 week. We'll listen to your feedback. We'll find the guests that you want me
113:59 We'll find the guests that you want me to speak to, and we'll continue to do
114:01 to speak to, and we'll continue to do what we do. Thank you so much.
114:08 [Music] Heat. Heat. N.
114:09 Heat. Heat. N. [Music]